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10.12688/f1000research.24441.2 http://scihub22266oqcxt.onion/10.12688/f1000research.24441.2 C7533733!7533733!33093945     free     free     free       F1000Res 2020 ; 9 (ä): ä Nephropedia Template TP {{tp|p=33093945|t=2020. Electrolyte Disturbances in SARS-CoV-2 Infection |pdf=|usr=}}{{33093945}} gab.com Text Electrolyte Disturbances in SARS-CoV-2 Infection JO: F1000Res http://www.kidney.de/pget.php?&tw=1&pmid=33093945 #FOAvip The global pandemic secondary to the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is leading to unprecedented global morbidity and mortality. With a bewildering array of complications, renal involvement in various forms is common, including serum electrolyte derangements. Hypokalaemia secondary to SARS-CoV-2 was common in a reported Chinese cohort. Here we review the emerging evidence on hypokalaemia and SARS-CoV-2 infection, the potential pathophysiological mechanisms based on early clinical and histopathological data and important clinical implications. Mechanisms of hypokalaemia are multifactorial and so the electrolyte disturbance can be difficult to avoid. We provide further support to the theory of renin-angiotensin-aldosterone (RAS) activation, discuss the strengths and weaknesses of implicating RAS involvement and highlight the importance of calculating the transtubular potassium gradient to identify those at risk of hypokalaemia and its complications. Twit Text FOAVip Electrolyte Disturbances in SARS-CoV-2 Infection ????F1000Res http://www.kidney.de/pget.php?&tw=1&pmid=33093945 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=33093945 #FOAvip English Wikipedia <ref>{{Cite pmid|33093945}}</ref> Electrolyte Disturbances in SARS-CoV-2 Infection #MMPMID33093945 Mabillard H; Sayer JA F1000Res 2020[]; 9 (ä): ä PMID33093945show ga The global pandemic secondary to the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is leading to unprecedented global morbidity and mortality. With a bewildering array of complications, renal involvement in various forms is common, including serum electrolyte derangements. Hypokalaemia secondary to SARS-CoV-2 was common in a reported Chinese cohort. Here we review the emerging evidence on hypokalaemia and SARS-CoV-2 infection, the potential pathophysiological mechanisms based on early clinical and histopathological data and important clinical implications. Mechanisms of hypokalaemia are multifactorial and so the electrolyte disturbance can be difficult to avoid. We provide further support to the theory of renin-angiotensin-aldosterone (RAS) activation, discuss the strengths and weaknesses of implicating RAS involvement and highlight the importance of calculating the transtubular potassium gradient to identify those at risk of hypokalaemia and its complications. äElectrolyte Disturbances in SARS-CoV-2 Infection (epmc).pdf DeepDyve Pubget Overpricing