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10.1093/ckj/sfaa099 http://scihub22266oqcxt.onion/10.1093/ckj/sfaa099 C7314187!7314187!32695326     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Kidney+J 2020 ; 13 (3): 354-61 Nephropedia Template TP {{tp|p=32695326|t=2020. Characterization of acute kidney injury in critically ill patients with severe coronavirus disease 2019 |pdf=|usr=}}{{32695326}} gab.com Text Characterization of acute kidney injury in critically ill patients with severe coronavirus disease 2019 JO: Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=32695326 #FOAvip Background: Coronavirus disease 2019 (COVID-19)-associated acute kidney injury (AKI) frequency, severity and characterization in critically ill patients has not been reported. Methods: Single-centre cohort performed from 3 March 2020 to 14 April 2020 in four intensive care units in Bordeaux University Hospital, France. All patients with COVID-19 and pulmonary severity criteria were included. AKI was defined using Kidney Disease: Improving Global Outcomes (KDIGO) criteria. A systematic urinary analysis was performed. The incidence, severity, clinical presentation, biological characterization (transient versus persistent AKI; proteinuria, haematuria and glycosuria) and short-term outcomes were evaluated. Results: Seventy-one patients were included, with basal serum creatinine (SCr) of 69?±?21?µmol/L. At admission, AKI was present in 8/71 (11%) patients. Median [interquartile range (IQR)] follow-up was 17 (12?23) days. AKI developed in a total of 57/71 (80%) patients, with 35% Stage 1, 35% Stage 2 and 30% Stage 3 AKI; 10/57 (18%) required renal replacement therapy (RRT). Transient AKI was present in only 4/55 (7%) patients and persistent AKI was observed in 51/55 (93%). Patients with persistent AKI developed a median (IQR) urine protein/creatinine of 82 (54?140) (mg/mmol) with an albuminuria/proteinuria ratio of 0.23?±?20, indicating predominant tubulointerstitial injury. Only two (4%) patients had glycosuria. At Day 7 after onset of AKI, six (11%) patients remained dependent on RRT, nine (16%) had SCr >200?µmol/L and four (7%) had died. Day 7 and Day 14 renal recovery occurred in 28% and 52%, respectively. Conclusion: Severe COVID-19-associated AKI is frequent, persistent, severe and characterized by an almost exclusive tubulointerstitial injury without glycosuria. Twit Text FOAVip Characterization of acute kidney injury in critically ill patients with severe coronavirus disease 2019 ????Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=32695326 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32695326 #FOAvip English Wikipedia <ref>{{Cite pmid|32695326}}</ref> Characterization of acute kidney injury in critically ill patients with severe coronavirus disease 2019 #MMPMID32695326 Rubin S; Orieux A; Prevel R; Garric A; Bats ML; Dabernat S; Camou F; Guisset O; Issa N; Mourissoux G; Dewitte A; Joannes-Boyau O; Fleureau C; Rozé H; Carrié C; Petit L; Clouzeau B; Sazio C; Bui HN; Pillet O; Rigothier C; Vargas F; Combe C; Gruson D; Boyer A Clin Kidney J 2020[Jun]; 13 (3): 354-61 PMID32695326show ga Background: Coronavirus disease 2019 (COVID-19)-associated acute kidney injury (AKI) frequency, severity and characterization in critically ill patients has not been reported. Methods: Single-centre cohort performed from 3 March 2020 to 14 April 2020 in four intensive care units in Bordeaux University Hospital, France. All patients with COVID-19 and pulmonary severity criteria were included. AKI was defined using Kidney Disease: Improving Global Outcomes (KDIGO) criteria. A systematic urinary analysis was performed. The incidence, severity, clinical presentation, biological characterization (transient versus persistent AKI; proteinuria, haematuria and glycosuria) and short-term outcomes were evaluated. Results: Seventy-one patients were included, with basal serum creatinine (SCr) of 69?±?21?µmol/L. At admission, AKI was present in 8/71 (11%) patients. Median [interquartile range (IQR)] follow-up was 17 (12?23) days. AKI developed in a total of 57/71 (80%) patients, with 35% Stage 1, 35% Stage 2 and 30% Stage 3 AKI; 10/57 (18%) required renal replacement therapy (RRT). Transient AKI was present in only 4/55 (7%) patients and persistent AKI was observed in 51/55 (93%). Patients with persistent AKI developed a median (IQR) urine protein/creatinine of 82 (54?140) (mg/mmol) with an albuminuria/proteinuria ratio of 0.23?±?20, indicating predominant tubulointerstitial injury. Only two (4%) patients had glycosuria. At Day 7 after onset of AKI, six (11%) patients remained dependent on RRT, nine (16%) had SCr >200?µmol/L and four (7%) had died. Day 7 and Day 14 renal recovery occurred in 28% and 52%, respectively. Conclusion: Severe COVID-19-associated AKI is frequent, persistent, severe and characterized by an almost exclusive tubulointerstitial injury without glycosuria. äCharacterization of acute kidney injury in critically ill patients wit(epmc).pdf DeepDyve Pubget Overpricing