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10.1183/13993003.01608-2020

http://scihub22266oqcxt.onion/10.1183/13993003.01608-2020
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C7301830!7301830!32554532
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suck abstract from ncbi


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pmid32554532      Eur+Respir+J 2020 ; 56 (1): ä
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  • Thrombosis and COVID-19 pneumonia: the clot thickens! #MMPMID32554532
  • Price LC; McCabe C; Garfield B; Wort SJ
  • Eur Respir J 2020[Jul]; 56 (1): ä PMID32554532show ga
  • At the end of last year, a novel coronavirus, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), resulted in an acute respiratory illness epidemic in Wuhan, China [1, 2]. The World Health Organization (WHO) termed this illness coronavirus disease 2019 (COVID-19). The coronavirus family have been shown to enter cells through binding angiotensin-converting enzyme 2 (ACE-2), found mainly on alveolar epithelium and endothelium. Activation of endothelial cells is thought to be the primary driver for the increasingly recognised complication of thrombosis.
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