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10.1002/ctm2.44 http://scihub22266oqcxt.onion/10.1002/ctm2.44 C7288983!7288983!32508062     free     free     free Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 261.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 261.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 261.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 261.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Transl+Med 2020 ; 10 (2): ä Nephropedia Template TP {{tp|p=32508062|t=2020. COVID?19 critical illness pathophysiology driven by diffuse pulmonary thrombi and pulmonary endothelial dysfunction responsive to thrombolysis |pdf=|usr=}}{{32508062}} gab.com Text COVID 19 critical illness pathophysiology driven by diffuse pulmonary thrombi and pulmonary endothelial dysfunction responsive to thrombolysis JO: Clin Transl Med http://www.kidney.de/pget.php?&tw=1&pmid=32508062 #FOAvip Patients with severe COVID?19 disease have been characterized as having the acute respiratory distress syndrome (ARDS). Critically ill COVID?19 patients have relatively well?preserved lung mechanics despite severe gas exchange abnormalities, a feature not consistent with classical ARDS but more consistent with pulmonary vascular disease. Many patients with severe COVID?19 also demonstrate markedly abnormal coagulation, with elevated d?dimers and higher rates of venous thromboembolism. We present four cases of patients with severe COVID?19 pneumonia with severe respiratory failure and shock, with evidence of markedly elevated dead?space ventilation who received tPA. All showed post treatment immediate improvements in gas exchange and/or hemodynamics. We suspect that severe COVID?19 pneumonia causes respiratory failure via pulmonary microthrombi and endothelial dysfunction. Treatment for COVID?19 pneumonia may warrant anticoagulation for milder cases and thrombolysis for more severe disease. Twit Text FOAVip COVID 19 critical illness pathophysiology driven by diffuse pulmonary thrombi and pulmonary endothelial dysfunction responsive to thrombolysis ????Clin Transl Med http://www.kidney.de/pget.php?&tw=1&pmid=32508062 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32508062 #FOAvip English Wikipedia <ref>{{Cite pmid|32508062}}</ref> COVID?19 critical illness pathophysiology driven by diffuse pulmonary thrombi and pulmonary endothelial dysfunction responsive to thrombolysis #MMPMID32508062 Poor HD; Ventetuolo CE; Tolbert T; Chun G; Serrao G; Zeidman A; Dangayach NS; Olin J; Kohli?Seth R; Powell CA Clin Transl Med 2020[Jun]; 10 (2): ä PMID32508062show ga Patients with severe COVID?19 disease have been characterized as having the acute respiratory distress syndrome (ARDS). Critically ill COVID?19 patients have relatively well?preserved lung mechanics despite severe gas exchange abnormalities, a feature not consistent with classical ARDS but more consistent with pulmonary vascular disease. Many patients with severe COVID?19 also demonstrate markedly abnormal coagulation, with elevated d?dimers and higher rates of venous thromboembolism. We present four cases of patients with severe COVID?19 pneumonia with severe respiratory failure and shock, with evidence of markedly elevated dead?space ventilation who received tPA. All showed post treatment immediate improvements in gas exchange and/or hemodynamics. We suspect that severe COVID?19 pneumonia causes respiratory failure via pulmonary microthrombi and endothelial dysfunction. Treatment for COVID?19 pneumonia may warrant anticoagulation for milder cases and thrombolysis for more severe disease. äCOVID?19 critical illness pathophysiology driven by diffuse pulmonary (epmc).pdf DeepDyve Pubget Overpricing