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10.1093/ehjcr/ytaa132

http://scihub22266oqcxt.onion/10.1093/ehjcr/ytaa132
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C7239209!7239209!33089039
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suck abstract from ncbi


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pmid33089039      Eur+Heart+J+Case+Rep 2020 ; 4 (FI1): 1-6
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  • Novel coronavirus 19 (COVID-19) associated sinus node dysfunction: a case series #MMPMID33089039
  • Peigh G; Leya MV; Baman JR; Cantey EP; Knight BP; Flaherty JD
  • Eur Heart J Case Rep 2020[Oct]; 4 (FI1): 1-6 PMID33089039show ga
  • Background: Novel coronavirus-19 disease (COVID-19) is associated with significant cardiovascular morbidity and mortality. To date, there have not been reports of sinus node dysfunction (SND) associated with COVID-19. This case series describes clinical characteristics, potential mechanisms, and short-term outcomes of COVID-19 patients who experience de novo SND. Case summary: We present two cases of new-onset SND in patients recently diagnosed with COVID-19. Patient 1 is a 70-year-old female with no major past medical history who was intubated for acute hypoxic respiratory failure secondary to COVID-19 pneumonia and developed new-onset sinus bradycardia without a compensatory increase in heart rate in response to relative hypotension. Patient 2 is an 81-year-old male with a past medical history of an ascending aortic aneurysm, hypertension, and obstructive sleep apnoea who required intubation for COVID-19-induced acute hypoxic respiratory failure and exhibited new-onset sinus bradycardia followed by numerous episodes of haemodynamically significant accelerated idioventricular rhythm. Two weeks following the onset of SND, both patients remain in sinus bradycardia. Discussion: COVID-19-associated SND has not previously been described. The potential mechanisms for SND in patients with COVID-19 include myocardial inflammation or direct viral infiltration. Patients diagnosed with COVID-19 should be monitored closely for the development of bradyarrhythmia and haemodynamic instability.
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