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10.1172/JCI131336 http://scihub22266oqcxt.onion/10.1172/JCI131336 C6763218!6763218!31483292     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Invest ä ; 129 (10): 4077-9 Nephropedia Template TP {{tp|p=31483292|t=ä. Less burn, more fat: electronic cigarettes and pulmonary lipid homeostasis |pdf=|usr=}}{{31483292}} gab.com Text Less burn, more fat: electronic cigarettes and pulmonary lipid homeostasis JO: J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=31483292 #FOAvip Electronic nicotine delivery systems (ENDS) are rapidly increasing in popularity due to the perception that they may represent a safe alternative to conventional cigarettes. However, a growing body of evidence indicates that ENDS exposure can disrupt maintenance of pulmonary immune homeostasis and antimicrobial immunity. In this issue of the JCI, Madison et al. demonstrate that in mice, chronic ENDS exposure induces profound alterations in lipid homeostasis. ENDS-exposed mice showed irregularities in the surfactant-secreting lamellar bodies within type 2 alveolar cells and increased intracellular phospholipid accumulation within alveolar macrophages. Moreover, ENDS-exposed mice displayed greater inflammation and tissue damage in response to influenza A, which may be due to downregulated expression of a viral pattern?recognition receptor in alveolar macrophages. Collectively, the results of this study identify previously unrecognized adverse effects of ENDS exposure on pulmonary lipid metabolism, although the implication of these effects on long-term respiratory health requires future exploration. Twit Text FOAVip Less burn, more fat: electronic cigarettes and pulmonary lipid homeostasis ????J Clin Invest http://www.kidney.de/pget.php?&tw=1&pmid=31483292 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=31483292 #FOAvip English Wikipedia <ref>{{Cite pmid|31483292}}</ref> Less burn, more fat: electronic cigarettes and pulmonary lipid homeostasis #MMPMID31483292 Singanayagam A; Snelgrove RJ J Clin Invest ä[]; 129 (10): 4077-9 PMID31483292show ga Electronic nicotine delivery systems (ENDS) are rapidly increasing in popularity due to the perception that they may represent a safe alternative to conventional cigarettes. However, a growing body of evidence indicates that ENDS exposure can disrupt maintenance of pulmonary immune homeostasis and antimicrobial immunity. In this issue of the JCI, Madison et al. demonstrate that in mice, chronic ENDS exposure induces profound alterations in lipid homeostasis. ENDS-exposed mice showed irregularities in the surfactant-secreting lamellar bodies within type 2 alveolar cells and increased intracellular phospholipid accumulation within alveolar macrophages. Moreover, ENDS-exposed mice displayed greater inflammation and tissue damage in response to influenza A, which may be due to downregulated expression of a viral pattern?recognition receptor in alveolar macrophages. Collectively, the results of this study identify previously unrecognized adverse effects of ENDS exposure on pulmonary lipid metabolism, although the implication of these effects on long-term respiratory health requires future exploration. äLess burn, more fat: electronic cigarettes and pulmonary lipid homeost(epmc).pdf DeepDyve Pubget Overpricing