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10.1073/pnas.1902310116 http://scihub22266oqcxt.onion/10.1073/pnas.1902310116 C6575592!6575592!31138703     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=31138703&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 265.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 265.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 265.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Proc+Natl+Acad+Sci+U+S+A 2019 ; 116 (24): 11888-93 Nephropedia Template TP {{tp|p=31138703|t=2019. BCL6 modulates tissue neutrophil survival and exacerbates pulmonary inflammation following influenza virus infection |pdf=|usr=}}{{31138703}} gab.com Text BCL6 modulates tissue neutrophil survival and exacerbates pulmonary inflammation following influenza virus infection JO: Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=31138703 #FOAvip Influenza A virus (IAV) infection poses a significant challenge to public health. The cellular and molecular mechanisms regulating the development of excessive pulmonary inflammation following IAV infection remain largely undefined. We find that the loss of BCL6 in neutrophils mitigates lung inflammation and promotes host resistance to IAV infection. The data provide mechanistic insight into how BCL6 regulates neutrophil-mediated lung inflammation through modulating neutrophil apoptosis at the site of IAV infection. Thus, targeting neutrophil survival in the tissue may be a promising therapeutic option to decrease host inflammation and minimize lung injury for severe IAV infection. Twit Text FOAVip BCL6 modulates tissue neutrophil survival and exacerbates pulmonary inflammation following influenza virus infection ????Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=31138703 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=31138703 #FOAvip English Wikipedia <ref>{{Cite pmid|31138703}}</ref> BCL6 modulates tissue neutrophil survival and exacerbates pulmonary inflammation following influenza virus infection #MMPMID31138703 Zhu B; Zhang R; Li C; Jiang L; Xiang M; Ye Z; Kita H; Melnick AM; Dent AL; Sun J Proc Natl Acad Sci U S A 2019[Jun]; 116 (24): 11888-93 PMID31138703show ga Influenza A virus (IAV) infection poses a significant challenge to public health. The cellular and molecular mechanisms regulating the development of excessive pulmonary inflammation following IAV infection remain largely undefined. We find that the loss of BCL6 in neutrophils mitigates lung inflammation and promotes host resistance to IAV infection. The data provide mechanistic insight into how BCL6 regulates neutrophil-mediated lung inflammation through modulating neutrophil apoptosis at the site of IAV infection. Thus, targeting neutrophil survival in the tissue may be a promising therapeutic option to decrease host inflammation and minimize lung injury for severe IAV infection. äBCL6 modulates tissue neutrophil survival and exacerbates pulmonary in(epmc).pdf DeepDyve Pubget Overpricing