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10.1016/j.celrep.2018.05.058 http://scihub22266oqcxt.onion/10.1016/j.celrep.2018.05.058 C6066340!6066340!29949755     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cell+Rep 2018 ; 23 (13): 3701-9 Nephropedia Template TP {{tp|p=29949755|t=2018. MOXI Is a Mitochondrial Micropeptide That Enhances Fatty Acid ?-Oxidation |pdf=|usr=}}{{29949755}} gab.com Text MOXI Is a Mitochondrial Micropeptide That Enhances Fatty Acid -Oxidation JO: Cell Rep http://www.kidney.de/pget.php?&tw=1&pmid=29949755 #FOAvip Micropeptide regulator of ?-oxidation (MOXI) is a conserved muscle-enriched protein encoded by an RNA transcript misannotated as non-coding. MOXI localizes to the inner mitochondrial membrane where it associates with the mitochondrial trifunctional protein, an enzyme complex that plays a critical role in fatty acid ?-oxidation. Isolated heart and skeletal muscle mitochondria from MOXI knockout mice exhibit a diminished ability to metabolize fatty acids, while transgenic MOXI overexpression leads to enhanced ?-oxidation. Additionally, hearts from MOXI knockout mice preferentially oxidize carbohydrates over fatty acids in an isolated perfused heart system compared to wild-type (WT) animals. MOXI knockout mice also exhibit a profound reduction in exercise capacity, highlighting the role of MOXI in metabolic control. The functional characterization of MOXI provides insight into the regulation of mitochondrial metabolism and energy homeostasis and underscores the regulatory potential of additional micropeptides that have yet to be identified. Twit Text FOAVip MOXI Is a Mitochondrial Micropeptide That Enhances Fatty Acid -Oxidation ????Cell Rep http://www.kidney.de/pget.php?&tw=1&pmid=29949755 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29949755 #FOAvip English Wikipedia <ref>{{Cite pmid|29949755}}</ref> MOXI Is a Mitochondrial Micropeptide That Enhances Fatty Acid ?-Oxidation #MMPMID29949755 Makarewich CA; Baskin KK; Munir AZ; Bezprozvannaya S; Sharma G; Khemtong C; Shah AM; McAnally JR; Malloy CR; Szweda LI; Bassel-Duby R; Olson EN Cell Rep 2018[Jun]; 23 (13): 3701-9 PMID29949755show ga Micropeptide regulator of ?-oxidation (MOXI) is a conserved muscle-enriched protein encoded by an RNA transcript misannotated as non-coding. MOXI localizes to the inner mitochondrial membrane where it associates with the mitochondrial trifunctional protein, an enzyme complex that plays a critical role in fatty acid ?-oxidation. Isolated heart and skeletal muscle mitochondria from MOXI knockout mice exhibit a diminished ability to metabolize fatty acids, while transgenic MOXI overexpression leads to enhanced ?-oxidation. Additionally, hearts from MOXI knockout mice preferentially oxidize carbohydrates over fatty acids in an isolated perfused heart system compared to wild-type (WT) animals. MOXI knockout mice also exhibit a profound reduction in exercise capacity, highlighting the role of MOXI in metabolic control. The functional characterization of MOXI provides insight into the regulation of mitochondrial metabolism and energy homeostasis and underscores the regulatory potential of additional micropeptides that have yet to be identified. äMOXI Is a Mitochondrial Micropeptide That Enhances Fatty Acid ?-Oxidat(epmc).pdf DeepDyve Pubget Overpricing