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suck abstract from ncbi


10.1016/j.jacbts.2017.11.006

http://scihub22266oqcxt.onion/10.1016/j.jacbts.2017.11.006
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C6060078!6060078 !30062205
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suck abstract from ncbi


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pmid30062205
      JACC+Basic+Transl+Sci 2018 ; 3 (2 ): 200-209
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  • Apolipoprotein A-I Reduces In-Stent Restenosis and Platelet Activation and Alters Neointimal Cellular Phenotype #MMPMID30062205
  • Vanags LZ ; Tan JTM ; Galougahi KK ; Schaefer A ; Wise SG ; Murphy A ; Ali ZA ; Bursill CA
  • JACC Basic Transl Sci 2018[Apr]; 3 (2 ): 200-209 PMID30062205 show ga
  • Even the most advanced drug-eluting stents evoke unresolved issues, including chronic inflammation, late thrombosis, and neoatherosclerosis. This highlights the need for novel strategies that improve stent biocompatibility. Our studies show that apolipoprotein A-I (apoA-I) reduces in-stent restenosis and platelet activation, and enhances endothelialization. These findings have therapeutic implications for improving stent biocompatibility.
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