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10.1038/labinvest.2017.112 http://scihub22266oqcxt.onion/10.1038/labinvest.2017.112 C6053075!6053075!29035376     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Lab+Invest 2018 ; 98 (2): 258-68 Nephropedia Template TP {{tp|p=29035376|t=2018. Oncogenic Epstein?Barr virus recruits Nm23-H1 to regulate chromatin modifiers |pdf=|usr=}}{{29035376}} gab.com Text Oncogenic Epstein Barr virus recruits Nm23-H1 to regulate chromatin modifiers JO: Lab Invest http://www.kidney.de/pget.php?&tw=1&pmid=29035376 #FOAvip In cancer progression, metastasis is a major cause of poor survival of patients and can be targeted for therapeutic interventions. The first discovered metastatic-suppressor Nm23-H1 possesses nucleoside diphosphate kinase, histidine kinase, and DNase activity as a broad-spectrum enzyme. Recent advances in cancer metastasis have opened new ways for the development of therapeutic molecular approaches. In this review, we provide a summary of the current understanding of Nm23/NDPKs in the context of viral oncogenesis. We also focused on Nm23-H1-mediated cellular events with an emphasis on chromatin modifications. How Nm23-H1 modulates the activities of chromatin modifiers through interaction with Epstein?Barr virus-encoded oncogenic antigens and related crosstalks are discussed in the context of other oncogenic viruses. We also described the current understanding of the cellular and viral interactions of Nm23-H1 and their reference to transcription regulation and metastasis. Further, we summarized the recent therapeutic approaches targeting Nm23 and its potential links to pathways that can be exploited by oncogenic viruses. Twit Text FOAVip Oncogenic Epstein Barr virus recruits Nm23-H1 to regulate chromatin modifiers ????Lab Invest http://www.kidney.de/pget.php?&tw=1&pmid=29035376 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29035376 #FOAvip English Wikipedia <ref>{{Cite pmid|29035376}}</ref> Oncogenic Epstein?Barr virus recruits Nm23-H1 to regulate chromatin modifiers #MMPMID29035376 Pandey S; Robertson ES Lab Invest 2018[Feb]; 98 (2): 258-68 PMID29035376show ga In cancer progression, metastasis is a major cause of poor survival of patients and can be targeted for therapeutic interventions. The first discovered metastatic-suppressor Nm23-H1 possesses nucleoside diphosphate kinase, histidine kinase, and DNase activity as a broad-spectrum enzyme. Recent advances in cancer metastasis have opened new ways for the development of therapeutic molecular approaches. In this review, we provide a summary of the current understanding of Nm23/NDPKs in the context of viral oncogenesis. We also focused on Nm23-H1-mediated cellular events with an emphasis on chromatin modifications. How Nm23-H1 modulates the activities of chromatin modifiers through interaction with Epstein?Barr virus-encoded oncogenic antigens and related crosstalks are discussed in the context of other oncogenic viruses. We also described the current understanding of the cellular and viral interactions of Nm23-H1 and their reference to transcription regulation and metastasis. Further, we summarized the recent therapeutic approaches targeting Nm23 and its potential links to pathways that can be exploited by oncogenic viruses. äOncogenic Epstein?Barr virus recruits Nm23-H1 to regulate chromatin mo(epmc).pdf DeepDyve Pubget Overpricing