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10.5173/ceju.2018.1583

http://scihub22266oqcxt.onion/10.5173/ceju.2018.1583
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C6051361!6051361!30038806
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suck abstract from ncbi

pmid30038806      Cent+European+J+Urol 2018 ; 71 (2): 168-76
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  • Molecular alterations associated with prostate cancer #MMPMID30038806
  • Garcia Perdomo HA; Zapata-Copete JA; Sanchez A
  • Cent European J Urol 2018[]; 71 (2): 168-76 PMID30038806show ga
  • Introduction: The amount of information and knowledge about pathways and genetic alterations regarding prostate cancer, including the tools available for its study has been recently increasing. Additionally, a variety of molecular signaling pathways control cell proliferation, however, this incompletely understood process is disturbed in cancer cells. Materials and methods: A literature review was made using the MEDLINE, Embase and LILACS databases searching for the following keywords: prostate neoplasms, prostate cancer, molecular medicine, genomics, pathways, and cell cycle. Results: Different biological mechanisms have been associated with the development of prostate cancer, such as alterations in tumor suppressor genes, oncogenes (TP53, RB1, among others) and CDKIs; DNA methylation; chromosomal alterations and rearrangements; changes in PTEN and PI3K / mTOR; global defects in apoptosis; alterations in the androgen receptor (AR); and epigenetic mechanisms. Conclusions: Good clinical practice and a practical approach have to be based on basic knowledge, thus, in this article, the main genetic alterations, mutations and pathways involved in prostate cancer development were reviewed.
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