Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1111/febs.14090 http://scihub22266oqcxt.onion/10.1111/febs.14090 C6049610!6049610!28444969     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       FEBS+J 2017 ; 284 (19): 3132-44 Nephropedia Template TP {{tp|p=28444969|t=2017. Metabolic reprogramming and epithelial-to-mesenchymal transition in cancer |pdf=|usr=}}{{28444969}} gab.com Text Metabolic reprogramming and epithelial-to-mesenchymal transition in cancer JO: FEBS J http://www.kidney.de/pget.php?&tw=1&pmid=28444969 #FOAvip Several lines of evidence indicate that during transformation epithelial cancer cells can acquire mesenchymal features via a process called epithelial-to-mesenchymal transition (EMT). This process endows cancer cells with increased invasive and migratory capacity, enabling tumour dissemination and metastasis. EMT is associated with a complex metabolic reprogramming, orchestrated by EMT transcription factors, which support the energy requirements of increased motility and growth in harsh environmental conditions. The discovery that mutations in metabolic genes such as FH, SDH and IDH activate EMT provided further evidence that EMT and metabolism are intertwined. In this review, we discuss the role of EMT in cancer and the underpinning metabolic reprogramming. We also put forward the hypothesis that, by altering chromatin structure and function, metabolic pathways engaged by EMT are necessary for its full activation. Twit Text FOAVip Metabolic reprogramming and epithelial-to-mesenchymal transition in cancer ????FEBS J http://www.kidney.de/pget.php?&tw=1&pmid=28444969 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28444969 #FOAvip English Wikipedia <ref>{{Cite pmid|28444969}}</ref> Metabolic reprogramming and epithelial-to-mesenchymal transition in cancer #MMPMID28444969 Sciacovelli M; Frezza C FEBS J 2017[Oct]; 284 (19): 3132-44 PMID28444969show ga Several lines of evidence indicate that during transformation epithelial cancer cells can acquire mesenchymal features via a process called epithelial-to-mesenchymal transition (EMT). This process endows cancer cells with increased invasive and migratory capacity, enabling tumour dissemination and metastasis. EMT is associated with a complex metabolic reprogramming, orchestrated by EMT transcription factors, which support the energy requirements of increased motility and growth in harsh environmental conditions. The discovery that mutations in metabolic genes such as FH, SDH and IDH activate EMT provided further evidence that EMT and metabolism are intertwined. In this review, we discuss the role of EMT in cancer and the underpinning metabolic reprogramming. We also put forward the hypothesis that, by altering chromatin structure and function, metabolic pathways engaged by EMT are necessary for its full activation. äMetabolic reprogramming and epithelial-to-mesenchymal transition in ca(epmc).pdf DeepDyve Pubget Overpricing