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10.1042/BST20160089 http://scihub22266oqcxt.onion/10.1042/BST20160089 C6042638!6042638!27528754     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Biochem+Soc+Trans 2016 ; 44 (4): 1073-8 Nephropedia Template TP {{tp|p=27528754|t=2016. Non-coding RNAs and disease: the classical ncRNAs make a comeback |pdf=|usr=}}{{27528754}} gab.com Text Non-coding RNAs and disease: the classical ncRNAs make a comeback JO: Biochem Soc Trans http://www.kidney.de/pget.php?&tw=1&pmid=27528754 #FOAvip Many human diseases have been attributed to mutation in the protein coding regions of the human genome. The protein coding portion of the human genome, however, is very small compared to the non-coding portion of the genome. As such, there are a disproportionate number of diseases attributed to the coding compared to the non-coding portion of the genome. It is now clear that the non-coding portion of the genome produces many functional non-coding RNAs and these RNAs are slowly being linked to human diseases. Here we discuss examples where mutation in classical non-coding RNAs have been attributed to human disease and identify the future potential for the non-coding portion of the genome in disease biology. Twit Text FOAVip Non-coding RNAs and disease: the classical ncRNAs make a comeback ????Biochem Soc Trans http://www.kidney.de/pget.php?&tw=1&pmid=27528754 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27528754 #FOAvip English Wikipedia <ref>{{Cite pmid|27528754}}</ref> Non-coding RNAs and disease: the classical ncRNAs make a comeback #MMPMID27528754 de Almeida RA; Fraczek MG; Parker S; Delneri D; O?Keefe RT Biochem Soc Trans 2016[Aug]; 44 (4): 1073-8 PMID27528754show ga Many human diseases have been attributed to mutation in the protein coding regions of the human genome. The protein coding portion of the human genome, however, is very small compared to the non-coding portion of the genome. As such, there are a disproportionate number of diseases attributed to the coding compared to the non-coding portion of the genome. It is now clear that the non-coding portion of the genome produces many functional non-coding RNAs and these RNAs are slowly being linked to human diseases. Here we discuss examples where mutation in classical non-coding RNAs have been attributed to human disease and identify the future potential for the non-coding portion of the genome in disease biology. äNon-coding RNAs and disease: the classical ncRNAs make a comeback (epmc).pdf DeepDyve Pubget Overpricing