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10.1155/2018/7565076 http://scihub22266oqcxt.onion/10.1155/2018/7565076 C6038472!6038472!30046303     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Gastroenterol+Res+Pract 2018 ; 2018 (ä): ä Nephropedia Template TP {{tp|p=30046303|t=2018. The Role of Autophagy and Related MicroRNAs in Inflammatory Bowel Disease |pdf=|usr=}}{{30046303}} gab.com Text The Role of Autophagy and Related MicroRNAs in Inflammatory Bowel Disease JO: Gastroenterol Res Pract http://www.kidney.de/pget.php?&tw=1&pmid=30046303 #FOAvip Accumulating evidence demonstrates that microRNA- (miR-) mediated posttranscriptional regulation plays an important role in autophagy in inflammatory bowel disease (IBD), a disease that is difficult to manage clinically because of the associated chronic recurrent nonspecific inflammation. Research indicates that microRNAs regulate autophagy via different pathways, playing an important role in the IBD process and providing a new perspective for IBD research. Related studies have shown that miR-142-3p, miR-320, miR-192, and miR-122 target NOD2, an IBD-relevant autophagy gene, to modulate autophagy in IBD. miR-142-3p, miR-93, miR-106B, miR-30C, miR-130a, miR-346, and miR-20a regulate autophagy by targeting ATG16L1 through several different pathways. miR-196 can downregulate IRGM and suppress autophagy by inhibiting the accumulation of LC3II. During the endoplasmic reticulum stress response, miR-665, miR-375, and miR-150 modulate autophagy by regulating the unfolded protein response, which may play an important role in IBD intestinal fibrosis. Regarding autophagy-related pathways, miR-146b, miR-221-5p, miR-132, miR-223, miR-155, and miR-21 regulate NF-?B or mTOR signaling to induce or inhibit autophagy in intestinal cells by releasing anti- or proinflammatory factors, respectively. Twit Text FOAVip The Role of Autophagy and Related MicroRNAs in Inflammatory Bowel Disease ????Gastroenterol Res Pract http://www.kidney.de/pget.php?&tw=1&pmid=30046303 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=30046303 #FOAvip English Wikipedia <ref>{{Cite pmid|30046303}}</ref> The Role of Autophagy and Related MicroRNAs in Inflammatory Bowel Disease #MMPMID30046303 Wang S; Huang Y; Zhou C; Wu H; Zhao J; Wu L; Zhao M; Zhang F; Liu H Gastroenterol Res Pract 2018[]; 2018 (ä): ä PMID30046303show ga Accumulating evidence demonstrates that microRNA- (miR-) mediated posttranscriptional regulation plays an important role in autophagy in inflammatory bowel disease (IBD), a disease that is difficult to manage clinically because of the associated chronic recurrent nonspecific inflammation. Research indicates that microRNAs regulate autophagy via different pathways, playing an important role in the IBD process and providing a new perspective for IBD research. Related studies have shown that miR-142-3p, miR-320, miR-192, and miR-122 target NOD2, an IBD-relevant autophagy gene, to modulate autophagy in IBD. miR-142-3p, miR-93, miR-106B, miR-30C, miR-130a, miR-346, and miR-20a regulate autophagy by targeting ATG16L1 through several different pathways. miR-196 can downregulate IRGM and suppress autophagy by inhibiting the accumulation of LC3II. During the endoplasmic reticulum stress response, miR-665, miR-375, and miR-150 modulate autophagy by regulating the unfolded protein response, which may play an important role in IBD intestinal fibrosis. Regarding autophagy-related pathways, miR-146b, miR-221-5p, miR-132, miR-223, miR-155, and miR-21 regulate NF-?B or mTOR signaling to induce or inhibit autophagy in intestinal cells by releasing anti- or proinflammatory factors, respectively. äThe Role of Autophagy and Related MicroRNAs in Inflammatory Bowel Dise(epmc).pdf DeepDyve Pubget Overpricing