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10.7150/ijms.24659

http://scihub22266oqcxt.onion/10.7150/ijms.24659
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C6036153!6036153!30013438
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suck abstract from ncbi


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pmid30013438      Int+J+Med+Sci 2018 ; 15 (10): 978-85
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  • Endothelial-cell inflammation and damage by reactive oxygen species are prevented by propofol via ABCA1-mediated cholesterol efflux #MMPMID30013438
  • Hsu CP; Lin CH; Kuo CY
  • Int J Med Sci 2018[]; 15 (10): 978-85 PMID30013438show ga
  • Background: Cholesterol efflux efficiency, reactive oxygen species, and inflammation are closely related to cardiovascular diseases. Our aim was to investigate the effect of propofol on cholesterol-loaded rat aortic endothelial cells after high-density lipoprotein treatment in vitro.Methods and Results: The results showed that propofol promoted cholesterol efflux and ameliorated inflammation and reactive oxygen species overproduction according to the analysis of p65 nuclear translocation and a 2?,7?-dichlorofluorescin diacetate assay, respectively.Conclusions: These results provide a possible explanation for the anti-inflammatory, antioxidant, and cholesterol efflux-promoting effects of propofol on rat aortic endothelial cells after incubation with high-density lipoprotein.
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