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10.5483/BMBRep.2018.51.6.109 http://scihub22266oqcxt.onion/10.5483/BMBRep.2018.51.6.109 C6033072!6033072!29764562     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       BMB+Rep 2018 ; 51 (6): 263-4 Nephropedia Template TP {{tp|p=29764562|t=2018. Osteoclast-derived SLIT3 is a coupling factor linking bone resorption to bone formation |pdf=|usr=}}{{29764562}} gab.com Text Osteoclast-derived SLIT3 is a coupling factor linking bone resorption to bone formation JO: BMB Rep http://www.kidney.de/pget.php?&tw=1&pmid=29764562 #FOAvip We identified osteoclast-derived SLIT3 as a new coupling factor using fractionated secretomics. Coupling links bone resorption to bone formation. SLIT3 stimulated the recruitment and proliferation of osteoblasts into bone remodeling sites via activation of ?-catenin. Autocrine signaling by SLIT3 also inhibited bone resorption by suppressing the fusion and differentiation of pre-osteoclasts. All mice lacking Slit3 or its receptor Robo1 showed an osteopenic phenotype with low bone formation and high bone resorption. A small truncated recombinant SLIT3 protein increased bone mass in an osteopenic mouse model. These results suggest that SLIT3 is a novel therapeutic target in metabolic bone diseases. Twit Text FOAVip Osteoclast-derived SLIT3 is a coupling factor linking bone resorption to bone formation ????BMB Rep http://www.kidney.de/pget.php?&tw=1&pmid=29764562 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29764562 #FOAvip English Wikipedia <ref>{{Cite pmid|29764562}}</ref> Osteoclast-derived SLIT3 is a coupling factor linking bone resorption to bone formation #MMPMID29764562 Koh JM BMB Rep 2018[Jun]; 51 (6): 263-4 PMID29764562show ga We identified osteoclast-derived SLIT3 as a new coupling factor using fractionated secretomics. Coupling links bone resorption to bone formation. SLIT3 stimulated the recruitment and proliferation of osteoblasts into bone remodeling sites via activation of ?-catenin. Autocrine signaling by SLIT3 also inhibited bone resorption by suppressing the fusion and differentiation of pre-osteoclasts. All mice lacking Slit3 or its receptor Robo1 showed an osteopenic phenotype with low bone formation and high bone resorption. A small truncated recombinant SLIT3 protein increased bone mass in an osteopenic mouse model. These results suggest that SLIT3 is a novel therapeutic target in metabolic bone diseases. äOsteoclast-derived SLIT3 is a coupling factor linking bone resorption (epmc).pdf DeepDyve Pubget Overpricing