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10.3390/ijms19061642 http://scihub22266oqcxt.onion/10.3390/ijms19061642 C6032371!6032371!29865171     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29865171&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2018 ; 19 (6): ä Nephropedia Template TP {{tp|p=29865171|t=2018. VEGF Upregulation in Viral Infections and Its Possible Therapeutic Implications |pdf=|usr=}}{{29865171}} gab.com Text VEGF Upregulation in Viral Infections and Its Possible Therapeutic Implications JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29865171 #FOAvip Several viruses are recognized as the direct or indirect causative agents of human tumors and other severe human diseases. Vascular endothelial growth factor (VEGF) is identified as a principal proangiogenic factor that enhances the production of new blood vessels from existing vascular network. Therefore, oncogenic viruses such as Kaposi?s sarcoma herpesvirus (KSHV) and Epstein-Barr virus (EBV) and non-oncogenic viruses such as herpes simplex virus (HSV-1) and dengue virus, which lack their own angiogenic factors, rely on the recruitment of cellular genes for angiogenesis in tumor progression or disease pathogenesis. This review summarizes how human viruses exploit the cellular signaling machinery to upregulate the expression of VEGF and benefit from its physiological functions for their own pathogenesis. Understanding the interplay between viruses and VEGF upregulation will pave the way to design targeted and effective therapeutic approaches for viral oncogenesis and severe diseases. Twit Text FOAVip VEGF Upregulation in Viral Infections and Its Possible Therapeutic Implications ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29865171 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29865171 #FOAvip English Wikipedia <ref>{{Cite pmid|29865171}}</ref> VEGF Upregulation in Viral Infections and Its Possible Therapeutic Implications #MMPMID29865171 Alkharsah KR Int J Mol Sci 2018[Jun]; 19 (6): ä PMID29865171show ga Several viruses are recognized as the direct or indirect causative agents of human tumors and other severe human diseases. Vascular endothelial growth factor (VEGF) is identified as a principal proangiogenic factor that enhances the production of new blood vessels from existing vascular network. Therefore, oncogenic viruses such as Kaposi?s sarcoma herpesvirus (KSHV) and Epstein-Barr virus (EBV) and non-oncogenic viruses such as herpes simplex virus (HSV-1) and dengue virus, which lack their own angiogenic factors, rely on the recruitment of cellular genes for angiogenesis in tumor progression or disease pathogenesis. This review summarizes how human viruses exploit the cellular signaling machinery to upregulate the expression of VEGF and benefit from its physiological functions for their own pathogenesis. Understanding the interplay between viruses and VEGF upregulation will pave the way to design targeted and effective therapeutic approaches for viral oncogenesis and severe diseases. äVEGF Upregulation in Viral Infections and Its Possible Therapeutic Imp(epmc).pdf DeepDyve Pubget Overpricing