Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1177/0300060518771265 http://scihub22266oqcxt.onion/10.1177/0300060518771265 C6023070!6023070!29708004     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Int+Med+Res 2018 ; 46 (6): 2170-6 Nephropedia Template TP {{tp|p=29708004|t=2018. Traumatic glioblastoma: commentary and suggested mechanism |pdf=|usr=}}{{29708004}} gab.com Text Traumatic glioblastoma: commentary and suggested mechanism JO: J Int Med Res http://www.kidney.de/pget.php?&tw=1&pmid=29708004 #FOAvip The role of head trauma in the development of glioblastoma is highly controversial and has been minimized since first put forward. This is not unexpected because skull injuries are overwhelmingly more common than glioblastoma. This paper presents a commentary based on the contributions of James Ewing, who established a major set of criteria for the recognition of an official relationship between trauma and cancer. Ewing?s criteria were very stringent. The scholars who succeeded Ewing have facilitated the characterization of traumatic brain injuries since the introduction of computed tomography and magnetic resonance imaging. Discussions of the various criteria that have since developed are now being conducted, and those of an unnecessarily limiting nature are being highlighted. Three transcription factors associated with traumatic brain injury have been identified: p53, hypoxia-inducible factor-1?, and c-MYC. A role for these three transcription factors in the relationship between traumatic brain injury and glioblastoma is suggested; this role may support a cause-and-effect link with the subsequent development of glioblastoma. Twit Text FOAVip Traumatic glioblastoma: commentary and suggested mechanism ????J Int Med Res http://www.kidney.de/pget.php?&tw=1&pmid=29708004 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29708004 #FOAvip English Wikipedia <ref>{{Cite pmid|29708004}}</ref> Traumatic glioblastoma: commentary and suggested mechanism #MMPMID29708004 Ohana N; Benharroch D; Sheinis D; Cohen A J Int Med Res 2018[Jun]; 46 (6): 2170-6 PMID29708004show ga The role of head trauma in the development of glioblastoma is highly controversial and has been minimized since first put forward. This is not unexpected because skull injuries are overwhelmingly more common than glioblastoma. This paper presents a commentary based on the contributions of James Ewing, who established a major set of criteria for the recognition of an official relationship between trauma and cancer. Ewing?s criteria were very stringent. The scholars who succeeded Ewing have facilitated the characterization of traumatic brain injuries since the introduction of computed tomography and magnetic resonance imaging. Discussions of the various criteria that have since developed are now being conducted, and those of an unnecessarily limiting nature are being highlighted. Three transcription factors associated with traumatic brain injury have been identified: p53, hypoxia-inducible factor-1?, and c-MYC. A role for these three transcription factors in the relationship between traumatic brain injury and glioblastoma is suggested; this role may support a cause-and-effect link with the subsequent development of glioblastoma. äTraumatic glioblastoma: commentary and suggested mechanism (epmc).pdf DeepDyve Pubget Overpricing