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10.1177/0300060517700299 http://scihub22266oqcxt.onion/10.1177/0300060517700299 C6023042!6023042!28459353     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Int+Med+Res 2018 ; 46 (6): 2141-8 Nephropedia Template TP {{tp|p=28459353|t=2018. Role of coagulopathy in glucocorticoid-induced osteonecrosis of the femoral head |pdf=|usr=}}{{28459353}} gab.com Text Role of coagulopathy in glucocorticoid-induced osteonecrosis of the femoral head JO: J Int Med Res http://www.kidney.de/pget.php?&tw=1&pmid=28459353 #FOAvip The two major theories of glucocorticoid (GC)-induced osteonecrosis of the femoral head (ONFH) are apoptosis and ischaemia. The traditional theory implicates ischaemia as the main aetiological factor because the final common pathway of ONFH is interruption of blood supply to the bone. The most common causes of interruption of blood supply include fat embolism and coagulation disorders. GCs can directly or indirectly lead to coagulation disorders, producing a hypercoagulable state, followed by poor blood flow, ischaemia, and eventually ONFH. This review summarizes the existing knowledge on coagulation disorders in the context of GC-induced ONFH, including hypofibrinolysis and thrombophilia, endothelial cell dysfunction and damage, endothelial cell apoptosis, lipid metabolism, platelet activation, and the effect of anticoagulant treatment. Twit Text FOAVip Role of coagulopathy in glucocorticoid-induced osteonecrosis of the femoral head ????J Int Med Res http://www.kidney.de/pget.php?&tw=1&pmid=28459353 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28459353 #FOAvip English Wikipedia <ref>{{Cite pmid|28459353}}</ref> Role of coagulopathy in glucocorticoid-induced osteonecrosis of the femoral head #MMPMID28459353 Zhang Q; L v J; Jin L J Int Med Res 2018[Jun]; 46 (6): 2141-8 PMID28459353show ga The two major theories of glucocorticoid (GC)-induced osteonecrosis of the femoral head (ONFH) are apoptosis and ischaemia. The traditional theory implicates ischaemia as the main aetiological factor because the final common pathway of ONFH is interruption of blood supply to the bone. The most common causes of interruption of blood supply include fat embolism and coagulation disorders. GCs can directly or indirectly lead to coagulation disorders, producing a hypercoagulable state, followed by poor blood flow, ischaemia, and eventually ONFH. This review summarizes the existing knowledge on coagulation disorders in the context of GC-induced ONFH, including hypofibrinolysis and thrombophilia, endothelial cell dysfunction and damage, endothelial cell apoptosis, lipid metabolism, platelet activation, and the effect of anticoagulant treatment. äRole of coagulopathy in glucocorticoid-induced osteonecrosis of the fe(epmc).pdf DeepDyve Pubget Overpricing