Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1111/apha.13066 http://scihub22266oqcxt.onion/10.1111/apha.13066 C5969253!5969253!29575542     free     free     free       Acta+Physiol+(Oxf) 2018 ; 223 (2): ä Nephropedia Template TP {{tp|p=29575542|t=2018. The stressed brain of humans and rodents |pdf=|usr=}}{{29575542}} gab.com Text The stressed brain of humans and rodents JO: Acta Physiol (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=29575542 #FOAvip After stress, the brain is exposed to waves of stress mediators, including corticosterone (in rodents) and cortisol (in humans). Corticosteroid hormones affect neuronal physiology in two time?domains: rapid, non?genomic actions primarily via mineralocorticoid receptors; and delayed genomic effects via glucocorticoid receptors. In parallel, cognitive processing is affected by stress hormones. Directly after stress, emotional behaviour involving the amygdala is strongly facilitated with cognitively a strong emphasis on the ?now? and ?self,? at the cost of higher cognitive processing. This enables the organism to quickly and adequately respond to the situation at hand. Several hours later, emotional circuits are dampened while functions related to the prefrontal cortex and hippocampus are promoted. This allows the individual to rationalize the stressful event and place it in the right context, which is beneficial in the long run. The brain's response to stress depends on an individual's genetic background in interaction with life events. Studies in rodents point to the possibility to prevent or reverse long?term consequences of early life adversity on cognitive processing, by normalizing the balance between the two receptor types for corticosteroid hormones at a critical moment just before the onset of puberty. Twit Text FOAVip The stressed brain of humans and rodents ????Acta Physiol (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=29575542 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29575542 #FOAvip English Wikipedia <ref>{{Cite pmid|29575542}}</ref> The stressed brain of humans and rodents #MMPMID29575542 Joëls M; Karst H; Sarabdjitsingh RA Acta Physiol (Oxf) 2018[Jun]; 223 (2): ä PMID29575542show ga After stress, the brain is exposed to waves of stress mediators, including corticosterone (in rodents) and cortisol (in humans). Corticosteroid hormones affect neuronal physiology in two time?domains: rapid, non?genomic actions primarily via mineralocorticoid receptors; and delayed genomic effects via glucocorticoid receptors. In parallel, cognitive processing is affected by stress hormones. Directly after stress, emotional behaviour involving the amygdala is strongly facilitated with cognitively a strong emphasis on the ?now? and ?self,? at the cost of higher cognitive processing. This enables the organism to quickly and adequately respond to the situation at hand. Several hours later, emotional circuits are dampened while functions related to the prefrontal cortex and hippocampus are promoted. This allows the individual to rationalize the stressful event and place it in the right context, which is beneficial in the long run. The brain's response to stress depends on an individual's genetic background in interaction with life events. Studies in rodents point to the possibility to prevent or reverse long?term consequences of early life adversity on cognitive processing, by normalizing the balance between the two receptor types for corticosteroid hormones at a critical moment just before the onset of puberty. äThe stressed brain of humans and rodents (epmc).pdf DeepDyve Pubget Overpricing