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10.1186/s12894-018-0360-5 http://scihub22266oqcxt.onion/10.1186/s12894-018-0360-5 C5956917!5956917!29769123     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       BMC+Urol 2018 ; 18 (ä): ä Nephropedia Template TP {{tp|p=29769123|t=2018. Obesity impairs male fertility through long-term effects on spermatogenesis |pdf=|usr=}}{{29769123}} gab.com Text Obesity impairs male fertility through long-term effects on spermatogenesis JO: BMC Urol http://www.kidney.de/pget.php?&tw=1&pmid=29769123 #FOAvip Objective: This study aimed to investigate the effect and possible underlying mechanisms of high-fat diet-induced obesity on spermatogenesis in male rats. Methods: A total of 45 male rats were randomly divided into control (n?=?15, normal diet) and obesity groups (n?=?30, high-fat diet) and were fed for 16 weeks. Body weight and organ indexes were determined after sacrifice. Indicators of reproductive function, including sperm count, sperm motility, apoptosis of spermatogenic cells, and oxidative stress levels, were measured. Serum metabolic parameters and reproductive hormones were also assayed. Results: Compared with the control group, epididymal sperm motility in the obese rats was significantly decreased (P?<?0.01). Morphological analysis of the obesity group showed vacuolar changes in seminiferous tubules, spermatogenic cell dysfunction, and increased apoptosis of spermatogenic cells in testicular tissue (P?<?0.05). The calculated free testosterone (cFT) concentration in serum was decreased (P?<?0.05), whereas the serum sex hormone-binding globulin (SHBG) level was significantly increased (P?<?0.01). The superoxide dismutase (SOD) concentration decreased and the malondialdehyde (MDA) concentration increased in testis tissues; however, neither changes were statistically significant (P?>?0.05). Results: Nutritional obesity can damage spermatogenesis in male rats due to long-term effects on spermatogenesis. Twit Text FOAVip Obesity impairs male fertility through long-term effects on spermatogenesis ????BMC Urol http://www.kidney.de/pget.php?&tw=1&pmid=29769123 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29769123 #FOAvip English Wikipedia <ref>{{Cite pmid|29769123}}</ref> Obesity impairs male fertility through long-term effects on spermatogenesis #MMPMID29769123 Jia YF; Feng Q; Ge ZY; Guo Y; Zhou F; Zhang KS; Wang XW; Lu WH; Liang XW; Gu YQ BMC Urol 2018[]; 18 (ä): ä PMID29769123show ga Objective: This study aimed to investigate the effect and possible underlying mechanisms of high-fat diet-induced obesity on spermatogenesis in male rats. Methods: A total of 45 male rats were randomly divided into control (n?=?15, normal diet) and obesity groups (n?=?30, high-fat diet) and were fed for 16 weeks. Body weight and organ indexes were determined after sacrifice. Indicators of reproductive function, including sperm count, sperm motility, apoptosis of spermatogenic cells, and oxidative stress levels, were measured. Serum metabolic parameters and reproductive hormones were also assayed. Results: Compared with the control group, epididymal sperm motility in the obese rats was significantly decreased (P??0.05). Results: Nutritional obesity can damage spermatogenesis in male rats due to long-term effects on spermatogenesis. äObesity impairs male fertility through long-term effects on spermatoge(epmc).pdf DeepDyve Pubget Overpricing