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10.1111/febs.14336 http://scihub22266oqcxt.onion/10.1111/febs.14336 C5947125!5947125!29151277     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       FEBS+J 2018 ; 285 (7): 1185-202 Nephropedia Template TP {{tp|p=29151277|t=2018. Mammalian mitophagy ? from in vitro molecules to in vivo models |pdf=|usr=}}{{29151277}} gab.com Text Mammalian mitophagy from in vitro molecules to in vivo models JO: FEBS J http://www.kidney.de/pget.php?&tw=1&pmid=29151277 #FOAvip The autophagic turnover of mitochondria, termed mitophagy, is thought to play an essential role in not only maintaining the health of the mitochondrial network but also that of the cell and organism as a whole. We have come a long way in identifying the molecular components required for mitophagy through extensive in vitro work and cell line characterisation, yet the physiological significance and context of these pathways remain largely unexplored. This is highlighted by the recent development of new mouse models that have revealed a striking level of variation in mitophagy, even under normal conditions. Here, we focus on programmed mitophagy and summarise our current understanding of why, how and where this takes place in mammals. Twit Text FOAVip Mammalian mitophagy from in vitro molecules to in vivo models ????FEBS J http://www.kidney.de/pget.php?&tw=1&pmid=29151277 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29151277 #FOAvip English Wikipedia <ref>{{Cite pmid|29151277}}</ref> Mammalian mitophagy ? from in vitro molecules to in vivo models #MMPMID29151277 Rodger CE; McWilliams TG; Ganley IG FEBS J 2018[Apr]; 285 (7): 1185-202 PMID29151277show ga The autophagic turnover of mitochondria, termed mitophagy, is thought to play an essential role in not only maintaining the health of the mitochondrial network but also that of the cell and organism as a whole. We have come a long way in identifying the molecular components required for mitophagy through extensive in vitro work and cell line characterisation, yet the physiological significance and context of these pathways remain largely unexplored. This is highlighted by the recent development of new mouse models that have revealed a striking level of variation in mitophagy, even under normal conditions. Here, we focus on programmed mitophagy and summarise our current understanding of why, how and where this takes place in mammals. äMammalian mitophagy ? from in vitro molecules to in vivo models (epmc).pdf DeepDyve Pubget Overpricing