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10.3389/fimmu.2018.00978 http://scihub22266oqcxt.onion/10.3389/fimmu.2018.00978 C5943566!5943566!29774034     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Immunol 2018 ; 9 (ä): ä Nephropedia Template TP {{tp|p=29774034|t=2018. Targeting VEGF/VEGFR to Modulate Antitumor Immunity |pdf=|usr=}}{{29774034}} gab.com Text Targeting VEGF/VEGFR to Modulate Antitumor Immunity JO: Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=29774034 #FOAvip In addition to the crucial role in promoting the growth of tumor vessels, vascular endothelial growth factor (VEGF) is also immunosuppressive. VEGF can inhibit the function of T cells, increase the recruitment of regulatory T cells (Tregs) and myeloid-derived suppressor cells (MDSCs), and hinder the differentiation and activation of dendritic cells (DCs). Recent studies have investigated the role of antiangiogenic agents in antitumor immunity, especially in recent 3 years. Therefore, it is necessary to update the role of targeting VEGF/VEGFR in antitumor immunity. In this review, we focus on the latest clinical and preclinical findings on the modulatory role of antiangiogenic agents targeting VEGF/VEGFR in immune cells, including effector T cells, Tregs, MDSCs, DCs, tumor-associated macrophages, and mast cells. Our review will be potentially helpful for the development of combinations of angiogenesis inhibitors with immunological modulators. Twit Text FOAVip Targeting VEGF/VEGFR to Modulate Antitumor Immunity ????Front Immunol http://www.kidney.de/pget.php?&tw=1&pmid=29774034 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29774034 #FOAvip English Wikipedia <ref>{{Cite pmid|29774034}}</ref> Targeting VEGF/VEGFR to Modulate Antitumor Immunity #MMPMID29774034 Yang J; Yan J; Liu B Front Immunol 2018[]; 9 (ä): ä PMID29774034show ga In addition to the crucial role in promoting the growth of tumor vessels, vascular endothelial growth factor (VEGF) is also immunosuppressive. VEGF can inhibit the function of T cells, increase the recruitment of regulatory T cells (Tregs) and myeloid-derived suppressor cells (MDSCs), and hinder the differentiation and activation of dendritic cells (DCs). Recent studies have investigated the role of antiangiogenic agents in antitumor immunity, especially in recent 3 years. Therefore, it is necessary to update the role of targeting VEGF/VEGFR in antitumor immunity. In this review, we focus on the latest clinical and preclinical findings on the modulatory role of antiangiogenic agents targeting VEGF/VEGFR in immune cells, including effector T cells, Tregs, MDSCs, DCs, tumor-associated macrophages, and mast cells. Our review will be potentially helpful for the development of combinations of angiogenesis inhibitors with immunological modulators. äTargeting VEGF/VEGFR to Modulate Antitumor Immunity (epmc).pdf DeepDyve Pubget Overpricing