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10.1093/jrr/rrx074 http://scihub22266oqcxt.onion/10.1093/jrr/rrx074 C5941153!5941153 !29281054     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29281054 &cmd=llinks): Failed to open stream: HTTP request failed! 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Radiation-induced unrepairable DSBs: their role in the late effects of radiation and possible applications to biodosimetry |pdf=|usr=}}{{29281054 }} gab.com Text Radiation-induced unrepairable DSBs: their role in the late effects of radiation and possible applications to biodosimetry JO: J Radiat Res http://www.kidney.de/pget.php?&tw=1&pmid=29281054 #FOAvip Although the vast majority of DNA damage induced by radiation exposure disappears rapidly, some lesions remain in the cell nucleus in very small quantities for days to months. These lesions may cause a considerable threat to an organism and include certain types of DNA double-strand breaks (DSBs) called 'unrepairable DSBs'. Unrepairable DSBs are thought to cause persistent malfunctioning of cells and tissues or cause late effects of radiation, especially the induction of delayed cell death, mutation, senescence, or carcinogenesis. Moreover, the measurement of unrepairable DSBs could potentially be used for retrospective biodosimetry or for identifying individuals at greater risk for developing the adverse effects associated with radiotherapy or chemotherapy. This review summarizes the concept of unrepairable DSBs in the context of persistent repair foci formed at DSBs. Twit Text FOAVip Radiation-induced unrepairable DSBs: their role in the late effects of radiation and possible applications to biodosimetry ????J Radiat Res http://www.kidney.de/pget.php?&tw=1&pmid=29281054 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29281054 #FOAvip English Wikipedia <ref>{{Cite pmid|29281054 }}</ref> Radiation-induced unrepairable DSBs: their role in the late effects of radiation and possible applications to biodosimetry #MMPMID29281054 Noda A J Radiat Res 2018[Apr]; 59 (suppl_2 ): ii114-ii120 PMID29281054 show ga Although the vast majority of DNA damage induced by radiation exposure disappears rapidly, some lesions remain in the cell nucleus in very small quantities for days to months. These lesions may cause a considerable threat to an organism and include certain types of DNA double-strand breaks (DSBs) called 'unrepairable DSBs'. Unrepairable DSBs are thought to cause persistent malfunctioning of cells and tissues or cause late effects of radiation, especially the induction of delayed cell death, mutation, senescence, or carcinogenesis. Moreover, the measurement of unrepairable DSBs could potentially be used for retrospective biodosimetry or for identifying individuals at greater risk for developing the adverse effects associated with radiotherapy or chemotherapy. This review summarizes the concept of unrepairable DSBs in the context of persistent repair foci formed at DSBs. |*Radiation [MESH] |*Radiometry [MESH] |Biomarkers/metabolism [MESH] |DNA Breaks, Double-Stranded/*radiation effects [MESH] |DNA Repair/*radiation effects [MESH] |Humans [MESH]Radiation-induced unrepairable DSBs: their role in the late effects of(epmc).pdf DeepDyve Pubget Overpricing