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10.3389/fmed.2018.00121 http://scihub22266oqcxt.onion/10.3389/fmed.2018.00121 C5936789!5936789!29761104     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Med+(Lausanne) 2018 ; 5 (ä): ä Nephropedia Template TP {{tp|p=29761104|t=2018. Platelets in Immune Response to Virus and Immunopathology of Viral Infections |pdf=|usr=}}{{29761104}} gab.com Text Platelets in Immune Response to Virus and Immunopathology of Viral Infections JO: Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=29761104 #FOAvip Platelets are essential effector cells in hemostasis. Aside from their role in coagulation, platelets are now recognized as major inflammatory cells with key roles in the innate and adaptive arms of the immune system. Activated platelets have key thromboinflammatory functions linking coagulation to immune responses in various infections, including in response to virus. Recent studies have revealed that platelets exhibit several pattern recognition receptors (PRR) including those from the toll-like receptor, NOD-like receptor, and C-type lectin receptor family and are first-line sentinels in detecting and responding to pathogens in the vasculature. Here, we review the main mechanisms of platelets interaction with viruses, including their ability to sustain viral infection and replication, their expression of specialized PRR, and activation of thromboinflammatory responses against viruses. Finally, we discuss the role of platelet-derived mediators and platelet interaction with vascular and immune cells in protective and pathophysiologic responses to dengue, influenza, and human immunodeficiency virus 1 infections. Twit Text FOAVip Platelets in Immune Response to Virus and Immunopathology of Viral Infections ????Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=29761104 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29761104 #FOAvip English Wikipedia <ref>{{Cite pmid|29761104}}</ref> Platelets in Immune Response to Virus and Immunopathology of Viral Infections #MMPMID29761104 Hottz ED; Bozza FA; Bozza PT Front Med (Lausanne) 2018[]; 5 (ä): ä PMID29761104show ga Platelets are essential effector cells in hemostasis. Aside from their role in coagulation, platelets are now recognized as major inflammatory cells with key roles in the innate and adaptive arms of the immune system. Activated platelets have key thromboinflammatory functions linking coagulation to immune responses in various infections, including in response to virus. Recent studies have revealed that platelets exhibit several pattern recognition receptors (PRR) including those from the toll-like receptor, NOD-like receptor, and C-type lectin receptor family and are first-line sentinels in detecting and responding to pathogens in the vasculature. Here, we review the main mechanisms of platelets interaction with viruses, including their ability to sustain viral infection and replication, their expression of specialized PRR, and activation of thromboinflammatory responses against viruses. Finally, we discuss the role of platelet-derived mediators and platelet interaction with vascular and immune cells in protective and pathophysiologic responses to dengue, influenza, and human immunodeficiency virus 1 infections. äPlatelets in Immune Response to Virus and Immunopathology of Viral Inf(epmc).pdf DeepDyve Pubget Overpricing