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10.1177/1073274817744658 http://scihub22266oqcxt.onion/10.1177/1073274817744658 C5933573!5933573!29353494     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cancer+Control 2018 ; 25 (1): ä Nephropedia Template TP {{tp|p=29353494|t=2018. Primary Liver Cancers, Part 2: Progression Pathways and Carcinogenesis |pdf=|usr=}}{{29353494}} gab.com Text Primary Liver Cancers, Part 2: Progression Pathways and Carcinogenesis JO: Cancer Control http://www.kidney.de/pget.php?&tw=1&pmid=29353494 #FOAvip Hepatocellular carcinoma (HCC) and primary intrahepatic cholangiocarcinoma (ICC) have been increasing in incidence worldwide and are leading causes of cancer death. Studies of the molecular alterations leading to these carcinomas provide insights into the key mechanisms involved. A literature review was conducted to identify articles with information relevant to current understanding of the etiologies and molecular pathogenesis of HCC and ICC. Chronic inflammatory diseases are the key etiological risk factors for both HCC and ICC, although other diseases play a role, and for many ICCs, an underlying risk factor is not identified. Mutations in catenin beta 1 (CTNBB1) and tumor protein 53 (P53) are the main genetic alterations in HCC. Isocitrate dehydrogenases 1 and 2 (IDH1/2), KRAS protooncogene GTPase (KRAS), a RAS Viral Oncogene Homolog in neoroblastoma (NRAS) and P53 are primary genetic alterations in ICC. In both diseases, the mutational landscape is dependent on the underlying etiology. The most significant etiologies and genetic processes involved in the carcinogenesis of HCC and ICC are reviewed. Twit Text FOAVip Primary Liver Cancers, Part 2: Progression Pathways and Carcinogenesis ????Cancer Control http://www.kidney.de/pget.php?&tw=1&pmid=29353494 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29353494 #FOAvip English Wikipedia <ref>{{Cite pmid|29353494}}</ref> Primary Liver Cancers, Part 2: Progression Pathways and Carcinogenesis #MMPMID29353494 Jiang K; Centeno BA Cancer Control 2018[Jan]; 25 (1): ä PMID29353494show ga Hepatocellular carcinoma (HCC) and primary intrahepatic cholangiocarcinoma (ICC) have been increasing in incidence worldwide and are leading causes of cancer death. Studies of the molecular alterations leading to these carcinomas provide insights into the key mechanisms involved. A literature review was conducted to identify articles with information relevant to current understanding of the etiologies and molecular pathogenesis of HCC and ICC. Chronic inflammatory diseases are the key etiological risk factors for both HCC and ICC, although other diseases play a role, and for many ICCs, an underlying risk factor is not identified. Mutations in catenin beta 1 (CTNBB1) and tumor protein 53 (P53) are the main genetic alterations in HCC. Isocitrate dehydrogenases 1 and 2 (IDH1/2), KRAS protooncogene GTPase (KRAS), a RAS Viral Oncogene Homolog in neoroblastoma (NRAS) and P53 are primary genetic alterations in ICC. In both diseases, the mutational landscape is dependent on the underlying etiology. The most significant etiologies and genetic processes involved in the carcinogenesis of HCC and ICC are reviewed. äPrimary Liver Cancers, Part 2: Progression Pathways and Carcinogenesis(epmc).pdf DeepDyve Pubget Overpricing