Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText.
Kick-your-searchterm to multiple Engines kick-your-query now !>
A dictionary by aggregated review articles of nephrology, medicine and the life sciences
Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

suck abstract from ncbi


10.3389/fimmu.2018.00838

http://scihub22266oqcxt.onion/10.3389/fimmu.2018.00838
suck pdf from google scholar
C5928262!5928262!29740443
unlimited free pdf from europmc29740443    free
PDF from PMC    free
html from PMC    free

suck abstract from ncbi

pmid29740443      Front+Immunol 2018 ; 9 (ä): ä
Nephropedia Template TP

gab.com Text

Twit Text FOAVip

Twit Text #

English Wikipedia


  • Endothelial Activation: The Ang/Tie Axis in Sepsis #MMPMID29740443
  • Leligdowicz A; Richard-Greenblatt M; Wright J; Crowley VM; Kain KC
  • Front Immunol 2018[]; 9 (ä): ä PMID29740443show ga
  • Sepsis, a dysregulated host response to infection that causes life-threatening organ dysfunction, is a highly heterogeneous syndrome with no specific treatment. Although sepsis can be caused by a wide variety of pathogenic organisms, endothelial dysfunction leading to vascular leak is a common mechanism of injury that contributes to the morbidity and mortality associated with the syndrome. Perturbations to the angiopoietin (Ang)/Tie2 axis cause endothelial cell activation and contribute to the pathogenesis of sepsis. In this review, we summarize how the Ang/Tie2 pathway is implicated in sepsis and describe its prognostic as well as therapeutic utility in life-threatening infections.
  • ä


  • DeepDyve
  • Pubget Overpricing
  • suck abstract from ncbi

    Linkout box