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10.1111/j.1349-7006.1991.tb01810.x http://scihub22266oqcxt.onion/10.1111/j.1349-7006.1991.tb01810.x C5918348!5918348 !1778762     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=1778762 &cmd=llinks): Failed to open stream: HTTP request failed! 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Possible application to medium-term organ bioassays for renal carcinogenesis modifiers in rats treated with N-ethyl-N-hydroxyethylnitrosamine and unilateral nephrectomy |pdf=|usr=}}{{1778762 }} gab.com Text Possible application to medium-term organ bioassays for renal carcinogenesis modifiers in rats treated with N-ethyl-N-hydroxyethylnitrosamine and unilateral nephrectomy JO: Jpn J Cancer Res http://www.kidney.de/pget.php?&tw=1&pmid=1778762 #FOAvip The effects of the renal tumor promoters; beta-cyclodextrin (beta-C), DL-serine (DL-S), basic lead acetate (LA), trisodium nitrilotriacetate monohydrate (NTA) and potassium bromate (KB), and diethylene glycol (DEG) as a negative control, on early stage of renal carcinogenesis were investigated in unilaterally nephrectomized male Wistar rats after N-ethyl-N-hydroxyethylnitrosamine (EHEN) administration. Wistar male rats were fed 1000 ppm EHEN diet for 2 weeks and the left kidney was removed at week 3, then the animals were divided into 7 groups of 15 rats each. These groups received the following treatments: 1000 ppm LA, 10000 ppm NTA or 500 ppm KB diet for 18 weeks from week 3; 45 mg/100 g body wt./day of beta-C injected sc for 7 days; 100 mg/100 g body wt. of DL-S injected sc biweekly for 6 weeks; 5% DEG in drinking water as a negative control for two days. Five rats in each group were killed at weeks 8, 12 and 20 and their kidneys were examined histologically. At week 20, the average numbers of adenomatous hyperplasias seen as preneoplastic lesions in the beta-C, DL-S, LA, NTA or KB groups were significantly higher than those in the DEG or control groups. Thus within a relatively short period of 20 weeks, promoting effects of chemicals can be detected as a significant increase of adenomatous hyperplasias in this model. Twit Text FOAVip Possible application to medium-term organ bioassays for renal carcinogenesis modifiers in rats treated with N-ethyl-N-hydroxyethylnitrosamine and unilateral nephrectomy ????Jpn J Cancer Res http://www.kidney.de/pget.php?&tw=1&pmid=1778762 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=1778762 #FOAvip English Wikipedia <ref>{{Cite pmid|1778762 }}</ref> Possible application to medium-term organ bioassays for renal carcinogenesis modifiers in rats treated with N-ethyl-N-hydroxyethylnitrosamine and unilateral nephrectomy #MMPMID1778762 Hiasa Y ; Konishi N ; Nakaoka S ; Nakamura M ; Nishii S ; Kitahori Y ; Ohshima M Jpn J Cancer Res 1991[Dec]; 82 (12 ): 1385-90 PMID1778762 show ga The effects of the renal tumor promoters; beta-cyclodextrin (beta-C), DL-serine (DL-S), basic lead acetate (LA), trisodium nitrilotriacetate monohydrate (NTA) and potassium bromate (KB), and diethylene glycol (DEG) as a negative control, on early stage of renal carcinogenesis were investigated in unilaterally nephrectomized male Wistar rats after N-ethyl-N-hydroxyethylnitrosamine (EHEN) administration. Wistar male rats were fed 1000 ppm EHEN diet for 2 weeks and the left kidney was removed at week 3, then the animals were divided into 7 groups of 15 rats each. These groups received the following treatments: 1000 ppm LA, 10000 ppm NTA or 500 ppm KB diet for 18 weeks from week 3; 45 mg/100 g body wt./day of beta-C injected sc for 7 days; 100 mg/100 g body wt. of DL-S injected sc biweekly for 6 weeks; 5% DEG in drinking water as a negative control for two days. Five rats in each group were killed at weeks 8, 12 and 20 and their kidneys were examined histologically. At week 20, the average numbers of adenomatous hyperplasias seen as preneoplastic lesions in the beta-C, DL-S, LA, NTA or KB groups were significantly higher than those in the DEG or control groups. Thus within a relatively short period of 20 weeks, promoting effects of chemicals can be detected as a significant increase of adenomatous hyperplasias in this model. |*Carcinogens [MESH] |*Diethylnitrosamine [MESH] |Animals [MESH] |Bromodeoxyuridine/metabolism [MESH] |Carcinogenicity Tests/methods [MESH] |Drug Synergism [MESH] |Hyperplasia [MESH] |Kidney Neoplasms/*chemically induced/metabolism/pathology [MESH] |Kidney/anatomy & histology/drug effects/pathology [MESH] |Male [MESH] |Nephrectomy/*adverse effects [MESH] |Organ Size/drug effects [MESH] |Rats [MESH] |Rats, Inbred Strains [MESH]Possible application to medium-term organ bioassays for renal carcinog(epmc).pdf DeepDyve Pubget Overpricing