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10.1146/annurev-physiol-022516-034348 http://scihub22266oqcxt.onion/10.1146/annurev-physiol-022516-034348 C5912892!5912892!27959619     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Annu+Rev+Physiol 2017 ; 79 (ä): 567-92 Nephropedia Template TP {{tp|p=27959619|t=2017. Macrophages and the Recovery from Acute and Chronic Inflammation |pdf=|usr=}}{{27959619}} gab.com Text Macrophages and the Recovery from Acute and Chronic Inflammation JO: Annu Rev Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27959619 #FOAvip In recent years, researchers have devoted much attention to the diverse roles of macrophages and their contributions to tissue development, wound healing, and angiogenesis. What should not be lost in the discussions regarding the diverse biology of these cells is that when perturbed, macrophages are the primary contributors to potentially pathological inflammatory processes. Macrophages stand poised to rapidly produce large amounts of inflammatory cytokines in response to danger signals. The production of these cytokines can initiate a cascade of inflammatory mediator release that can lead to wholesale tissue destruction. The destructive inflammatory capability of macrophages is amplified by exposure to exogenous interferon-?, which prolongs and heightens inflammatory responses. In simple terms, macrophages can thus be viewed as incendiary devices with hair triggers waiting to detonate. We have begun to ask questions about how these cells can be regulated to mitigate the collateral destruction associated with macrophage activation. Twit Text FOAVip Macrophages and the Recovery from Acute and Chronic Inflammation ????Annu Rev Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27959619 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27959619 #FOAvip English Wikipedia <ref>{{Cite pmid|27959619}}</ref> Macrophages and the Recovery from Acute and Chronic Inflammation #MMPMID27959619 Hamidzadeh K; Christensen SM; Dalby E; Chandrasekaran P; Mosser DM Annu Rev Physiol 2017[Feb]; 79 (ä): 567-92 PMID27959619show ga In recent years, researchers have devoted much attention to the diverse roles of macrophages and their contributions to tissue development, wound healing, and angiogenesis. What should not be lost in the discussions regarding the diverse biology of these cells is that when perturbed, macrophages are the primary contributors to potentially pathological inflammatory processes. Macrophages stand poised to rapidly produce large amounts of inflammatory cytokines in response to danger signals. The production of these cytokines can initiate a cascade of inflammatory mediator release that can lead to wholesale tissue destruction. The destructive inflammatory capability of macrophages is amplified by exposure to exogenous interferon-?, which prolongs and heightens inflammatory responses. In simple terms, macrophages can thus be viewed as incendiary devices with hair triggers waiting to detonate. We have begun to ask questions about how these cells can be regulated to mitigate the collateral destruction associated with macrophage activation. äMacrophages and the Recovery from Acute and Chronic Inflammation (epmc).pdf DeepDyve Pubget Overpricing