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10.1074/jbc.R117.810366 http://scihub22266oqcxt.onion/10.1074/jbc.R117.810366 C5900756!5900756!29371398     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Biol+Chem 2018 ; 293 (15): 5386-95 Nephropedia Template TP {{tp|p=29371398|t=2018. A molecular perspective of mammalian autophagosome biogenesis |pdf=|usr=}}{{29371398}} gab.com Text A molecular perspective of mammalian autophagosome biogenesis JO: J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=29371398 #FOAvip Autophagy is a highly conserved process and is essential for the maintenance of cellular homeostasis. Autophagy occurs at a basal level in all cells, but it can be up-regulated during stress, starvation, or infection. Misregulation of autophagy has been linked to various disorders, including cancer, neurodegeneration, and immune diseases. Here, we discuss the essential proteins acting in the formation of an autophagosome, with a focus on the ULK and VPS34 kinase complexes, phosphatidylinositol 3-phosphate effector proteins, and the transmembrane autophagy-related protein ATG9. The function and regulation of these and other autophagy-related proteins acting during formation will be addressed, in particular during amino acid starvation. Twit Text FOAVip A molecular perspective of mammalian autophagosome biogenesis ????J Biol Chem http://www.kidney.de/pget.php?&tw=1&pmid=29371398 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29371398 #FOAvip English Wikipedia <ref>{{Cite pmid|29371398}}</ref> A molecular perspective of mammalian autophagosome biogenesis #MMPMID29371398 Mercer TJ; Gubas A; Tooze SA J Biol Chem 2018[Apr]; 293 (15): 5386-95 PMID29371398show ga Autophagy is a highly conserved process and is essential for the maintenance of cellular homeostasis. Autophagy occurs at a basal level in all cells, but it can be up-regulated during stress, starvation, or infection. Misregulation of autophagy has been linked to various disorders, including cancer, neurodegeneration, and immune diseases. Here, we discuss the essential proteins acting in the formation of an autophagosome, with a focus on the ULK and VPS34 kinase complexes, phosphatidylinositol 3-phosphate effector proteins, and the transmembrane autophagy-related protein ATG9. The function and regulation of these and other autophagy-related proteins acting during formation will be addressed, in particular during amino acid starvation. äA molecular perspective of mammalian autophagosome biogenesis (epmc).pdf DeepDyve Pubget Overpricing