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10.7555/JBR.31.20160124 http://scihub22266oqcxt.onion/10.7555/JBR.31.20160124 C5895572!5895572!28546516     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 253.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Biomed+Res 2018 ; 32 (2): 81-90 Nephropedia Template TP {{tp|p=28546516|t=2018. Re-evaluating the role of epithelial-mesenchymal-transition in cancer progression |pdf=|usr=}}{{28546516}} gab.com Text Re-evaluating the role of epithelial-mesenchymal-transition in cancer progression JO: J Biomed Res http://www.kidney.de/pget.php?&tw=1&pmid=28546516 #FOAvip Epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) are essential for embryonic development and also important in cancer progression. In a conventional model, epithelial-like cancer cells transit to mesenchymal-like tumor cells with great motility via EMT transcription factors; these mesenchymal-like cells migrate through the circulation system, relocate to a suitable site and then convert back to an epithelial-like phenotype to regenerate the tumor. However, recent findings challenge this conventional model and support the existence of a stable hybrid epithelial/mesenchymal (E/M) tumor population. Hybrid E/M tumor cells exhibit both epithelial and mesenchymal properties, possess great metastatic and tumorigenic capacity and are associated with poorer patient prognosis. The hybrid E/M model and associated regulatory networks represent a conceptual change regarding tumor metastasis and organ colonization. It may lead to the development of novel treatment strategies to ultimately stop cancer progression and improve disease-free survival. Twit Text FOAVip Re-evaluating the role of epithelial-mesenchymal-transition in cancer progression ????J Biomed Res http://www.kidney.de/pget.php?&tw=1&pmid=28546516 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28546516 #FOAvip English Wikipedia <ref>{{Cite pmid|28546516}}</ref> Re-evaluating the role of epithelial-mesenchymal-transition in cancer progression #MMPMID28546516 Sulaiman A; Yao Z; Wang L J Biomed Res 2018[Mar]; 32 (2): 81-90 PMID28546516show ga Epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) are essential for embryonic development and also important in cancer progression. In a conventional model, epithelial-like cancer cells transit to mesenchymal-like tumor cells with great motility via EMT transcription factors; these mesenchymal-like cells migrate through the circulation system, relocate to a suitable site and then convert back to an epithelial-like phenotype to regenerate the tumor. However, recent findings challenge this conventional model and support the existence of a stable hybrid epithelial/mesenchymal (E/M) tumor population. Hybrid E/M tumor cells exhibit both epithelial and mesenchymal properties, possess great metastatic and tumorigenic capacity and are associated with poorer patient prognosis. The hybrid E/M model and associated regulatory networks represent a conceptual change regarding tumor metastasis and organ colonization. It may lead to the development of novel treatment strategies to ultimately stop cancer progression and improve disease-free survival. äRe-evaluating the role of epithelial-mesenchymal-transition in cancer (epmc).pdf DeepDyve Pubget Overpricing