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10.3727/105221617X15042750874156 http://scihub22266oqcxt.onion/10.3727/105221617X15042750874156 C5885149!5885149!28893351     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Gene+Expr 2017 ; 17 (4): 277-87 Nephropedia Template TP {{tp|p=28893351|t=2017. Hepatic Ischemia/Reperfusion: Mechanisms of Tissue Injury, Repair, and Regeneration |pdf=|usr=}}{{28893351}} gab.com Text Hepatic Ischemia/Reperfusion: Mechanisms of Tissue Injury, Repair, and Regeneration JO: Gene Expr http://www.kidney.de/pget.php?&tw=1&pmid=28893351 #FOAvip Hepatic ischemia/reperfusion (I/R) injury is a major complication of liver surgery, including liver resection, liver transplantation, and trauma surgery. Much has been learned about the inflammatory injury response induced by I/R, including the cascade of proinflammatory mediators and recruitment of activated leukocytes. In this review, we discuss the complex network of events that culminate in liver injury after I/R, including cellular, protein, and molecular mechanisms. In addition, we address the known endogenous regulatory mediators that function to maintain homeostasis and resolve injury. Finally, we cover more recent insights into how the liver repairs and regenerates after I/R injury, a setting in which physical mass remains unchanged, but functional liver mass is greatly reduced. In this regard, we focus on recent work highlighting a novel role of CXC chemokines as important regulators of hepatocyte proliferation and liver regeneration after I/R injury. Twit Text FOAVip Hepatic Ischemia/Reperfusion: Mechanisms of Tissue Injury, Repair, and Regeneration ????Gene Expr http://www.kidney.de/pget.php?&tw=1&pmid=28893351 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28893351 #FOAvip English Wikipedia <ref>{{Cite pmid|28893351}}</ref> Hepatic Ischemia/Reperfusion: Mechanisms of Tissue Injury, Repair, and Regeneration #MMPMID28893351 Konishi T; Lentsch AB Gene Expr 2017[]; 17 (4): 277-87 PMID28893351show ga Hepatic ischemia/reperfusion (I/R) injury is a major complication of liver surgery, including liver resection, liver transplantation, and trauma surgery. Much has been learned about the inflammatory injury response induced by I/R, including the cascade of proinflammatory mediators and recruitment of activated leukocytes. In this review, we discuss the complex network of events that culminate in liver injury after I/R, including cellular, protein, and molecular mechanisms. In addition, we address the known endogenous regulatory mediators that function to maintain homeostasis and resolve injury. Finally, we cover more recent insights into how the liver repairs and regenerates after I/R injury, a setting in which physical mass remains unchanged, but functional liver mass is greatly reduced. In this regard, we focus on recent work highlighting a novel role of CXC chemokines as important regulators of hepatocyte proliferation and liver regeneration after I/R injury. äHepatic Ischemia/Reperfusion: Mechanisms of Tissue Injury, Repair, and(epmc).pdf DeepDyve Pubget Overpricing