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10.3390/ijms19030730 http://scihub22266oqcxt.onion/10.3390/ijms19030730 C5877591!5877591!29510522     free     free     free       Int+J+Mol+Sci 2018 ; 19 (3): ä Nephropedia Template TP {{tp|p=29510522|t=2018. The Balance of Th17 versus Treg Cells in Autoimmunity |pdf=|usr=}}{{29510522}} gab.com Text The Balance of Th17 versus Treg Cells in Autoimmunity JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29510522 #FOAvip T helper type 17 (Th17) cells and pTreg cells, which share a common precursor cell (the naïve CD4 T cell), require a common tumor growth factor (TGF)-? signal for initial differentiation. However, terminally differentiated cells fulfill opposite functions: Th17 cells cause autoimmunity and inflammation, whereas Treg cells inhibit these phenomena and maintain immune homeostasis. Thus, unraveling the mechanisms that affect the Th17/Treg cell balance is critical if we are to better understand autoimmunity and tolerance. Recent studies have identified many factors that influence this balance; these factors range from signaling pathways triggered by T cell receptors, costimulatory receptors, and cytokines, to various metabolic pathways and the intestinal microbiota. This review article summarizes recent advances in our understanding of the Th17/Treg balance and its implications with respect to autoimmune disease. Twit Text FOAVip The Balance of Th17 versus Treg Cells in Autoimmunity ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29510522 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29510522 #FOAvip English Wikipedia <ref>{{Cite pmid|29510522}}</ref> The Balance of Th17 versus Treg Cells in Autoimmunity #MMPMID29510522 Lee GR Int J Mol Sci 2018[Mar]; 19 (3): ä PMID29510522show ga T helper type 17 (Th17) cells and pTreg cells, which share a common precursor cell (the naïve CD4 T cell), require a common tumor growth factor (TGF)-? signal for initial differentiation. However, terminally differentiated cells fulfill opposite functions: Th17 cells cause autoimmunity and inflammation, whereas Treg cells inhibit these phenomena and maintain immune homeostasis. Thus, unraveling the mechanisms that affect the Th17/Treg cell balance is critical if we are to better understand autoimmunity and tolerance. Recent studies have identified many factors that influence this balance; these factors range from signaling pathways triggered by T cell receptors, costimulatory receptors, and cytokines, to various metabolic pathways and the intestinal microbiota. This review article summarizes recent advances in our understanding of the Th17/Treg balance and its implications with respect to autoimmune disease. äThe Balance of Th17 versus Treg Cells in Autoimmunity (epmc).pdf DeepDyve Pubget Overpricing