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10.1186/s12931-018-0756-5 http://scihub22266oqcxt.onion/10.1186/s12931-018-0756-5 C5872399!5872399!29587748     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29587748&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Respir+Res 2018 ; 19 (ä): ä Nephropedia Template TP {{tp|p=29587748|t=2018. Regulation of alveolar macrophage death in acute lung inflammation |pdf=|usr=}}{{29587748}} gab.com Text Regulation of alveolar macrophage death in acute lung inflammation JO: Respir Res http://www.kidney.de/pget.php?&tw=1&pmid=29587748 #FOAvip Acute lung injury (ALI) and its severe form, known as acute respiratory distress syndrome (ARDS), are caused by direct pulmonary insults and indirect systemic inflammatory responses that result from conditions such as sepsis, trauma, and major surgery. The reciprocal influences between pulmonary and systemic inflammation augments the inflammatory process in the lung and promotes the development of ALI. Emerging evidence has revealed that alveolar macrophage (AM) death plays important roles in the progression of lung inflammation through its influence on other immune cell populations in the lung. Cell death and tissue inflammation form a positive feedback cycle, ultimately leading to exaggerated inflammation and development of disease. Pharmacological manipulation of AM death signals may serve as a logical therapeutic strategy for ALI/ARDS. This review will focus on recent advances in the regulation and underlying mechanisms of AM death as well as the influence of AM death on the development of ALI. Twit Text FOAVip Regulation of alveolar macrophage death in acute lung inflammation ????Respir Res http://www.kidney.de/pget.php?&tw=1&pmid=29587748 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29587748 #FOAvip English Wikipedia <ref>{{Cite pmid|29587748}}</ref> Regulation of alveolar macrophage death in acute lung inflammation #MMPMID29587748 Fan EKY; Fan J Respir Res 2018[]; 19 (ä): ä PMID29587748show ga Acute lung injury (ALI) and its severe form, known as acute respiratory distress syndrome (ARDS), are caused by direct pulmonary insults and indirect systemic inflammatory responses that result from conditions such as sepsis, trauma, and major surgery. The reciprocal influences between pulmonary and systemic inflammation augments the inflammatory process in the lung and promotes the development of ALI. Emerging evidence has revealed that alveolar macrophage (AM) death plays important roles in the progression of lung inflammation through its influence on other immune cell populations in the lung. Cell death and tissue inflammation form a positive feedback cycle, ultimately leading to exaggerated inflammation and development of disease. Pharmacological manipulation of AM death signals may serve as a logical therapeutic strategy for ALI/ARDS. This review will focus on recent advances in the regulation and underlying mechanisms of AM death as well as the influence of AM death on the development of ALI. äRegulation of alveolar macrophage death in acute lung inflammation (epmc).pdf DeepDyve Pubget Overpricing