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10.1038/icb.2016.113 http://scihub22266oqcxt.onion/10.1038/icb.2016.113 C5857356!5857356!27826146     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Immunol+Cell+Biol 2017 ; 95 (2): 146-51 Nephropedia Template TP {{tp|p=27826146|t=2017. A motive for killing: effector functions of regulated lytic cell death |pdf=|usr=}}{{27826146}} gab.com Text A motive for killing: effector functions of regulated lytic cell death JO: Immunol Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=27826146 #FOAvip Immunological responses activated by pathogen recognition come in many guises. The proliferation, differentiation and recruitment of immune cells, and the production of inflammatory cytokines and chemokines are central to lifelong immunity. Cell death serves as a key function in the resolution of innate and adaptive immune responses. It also coordinates cell-intrinsic effector functions to restrict infection. Necrosis was formally considered a passive form of cell death or a consequence of pathogen virulence factor expression, and necrotic tissue is frequently associated with infection. However, there is now emerging evidence that points to a role for regulated forms of necrosis, such as pyroptosis and necroptosis, driving inflammation and shaping the immune response. Twit Text FOAVip A motive for killing: effector functions of regulated lytic cell death ????Immunol Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=27826146 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27826146 #FOAvip English Wikipedia <ref>{{Cite pmid|27826146}}</ref> A motive for killing: effector functions of regulated lytic cell death #MMPMID27826146 Bliss-Moreau M; Chen AA; D?Cruz A; Croker BA Immunol Cell Biol 2017[Feb]; 95 (2): 146-51 PMID27826146show ga Immunological responses activated by pathogen recognition come in many guises. The proliferation, differentiation and recruitment of immune cells, and the production of inflammatory cytokines and chemokines are central to lifelong immunity. Cell death serves as a key function in the resolution of innate and adaptive immune responses. It also coordinates cell-intrinsic effector functions to restrict infection. Necrosis was formally considered a passive form of cell death or a consequence of pathogen virulence factor expression, and necrotic tissue is frequently associated with infection. However, there is now emerging evidence that points to a role for regulated forms of necrosis, such as pyroptosis and necroptosis, driving inflammation and shaping the immune response. äA motive for killing: effector functions of regulated lytic cell death(epmc).pdf DeepDyve Pubget Overpricing