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10.3390/ijms19020507 http://scihub22266oqcxt.onion/10.3390/ijms19020507 C5855729!5855729!29419744     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Int+J+Mol+Sci 2018 ; 19 (2): ä Nephropedia Template TP {{tp|p=29419744|t=2018. Extracellular Matrix Metalloproteinase Inducer EMMPRIN (CD147) in Cardiovascular Disease |pdf=|usr=}}{{29419744}} gab.com Text Extracellular Matrix Metalloproteinase Inducer EMMPRIN (CD147) in Cardiovascular Disease JO: Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29419744 #FOAvip The receptor EMMPRIN is involved in the development and progression of cardiovascular diseases and in the pathogenesis of myocardial infarction. There are several binding partners of EMMPRIN mediating the effects of EMMPRIN in cardiovascular diseases. EMMPRIN interaction with most binding partners leads to disease progression by mediating cytokine or chemokine release, the activation of platelets and monocytes, as well as the formation of monocyte-platelet aggregates (MPAs). EMMPRIN is also involved in atherosclerosis by mediating the infiltration of pro-inflammatory cells. There is also evidence that EMMPRIN controls energy metabolism of cells and that EMMPRIN binding partners modulate intracellular glycosylation and trafficking of EMMPRIN towards the cell membrane. In this review, we systematically discuss these multifaceted roles of EMMPRIN and its interaction partners, such as Cyclophilins, in cardiovascular disease. Twit Text FOAVip Extracellular Matrix Metalloproteinase Inducer EMMPRIN (CD147) in Cardiovascular Disease ????Int J Mol Sci http://www.kidney.de/pget.php?&tw=1&pmid=29419744 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29419744 #FOAvip English Wikipedia <ref>{{Cite pmid|29419744}}</ref> Extracellular Matrix Metalloproteinase Inducer EMMPRIN (CD147) in Cardiovascular Disease #MMPMID29419744 von Ungern-Sternberg SNI; Zernecke A; Seizer P Int J Mol Sci 2018[Feb]; 19 (2): ä PMID29419744show ga The receptor EMMPRIN is involved in the development and progression of cardiovascular diseases and in the pathogenesis of myocardial infarction. There are several binding partners of EMMPRIN mediating the effects of EMMPRIN in cardiovascular diseases. EMMPRIN interaction with most binding partners leads to disease progression by mediating cytokine or chemokine release, the activation of platelets and monocytes, as well as the formation of monocyte-platelet aggregates (MPAs). EMMPRIN is also involved in atherosclerosis by mediating the infiltration of pro-inflammatory cells. There is also evidence that EMMPRIN controls energy metabolism of cells and that EMMPRIN binding partners modulate intracellular glycosylation and trafficking of EMMPRIN towards the cell membrane. In this review, we systematically discuss these multifaceted roles of EMMPRIN and its interaction partners, such as Cyclophilins, in cardiovascular disease. äExtracellular Matrix Metalloproteinase Inducer EMMPRIN (CD147) in Card(epmc).pdf DeepDyve Pubget Overpricing