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http://scihub22266oqcxt.onion/ C5847309!5847309!29560368     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Med+Hypothesis+Discov+Innov+Ophthalmol 2017 ; 6 (4): 136-42 Nephropedia Template TP {{tp|p=29560368|t=2017. Surgical Versus Medical Treatment for Diabetic Macular Edema: A Review |pdf=|usr=}}{{29560368}} gab.com Text Surgical Versus Medical Treatment for Diabetic Macular Edema: A Review JO: Med Hypothesis Discov Innov Ophthalmol http://www.kidney.de/pget.php?&tw=1&pmid=29560368 #FOAvip We aimed to compare the results of pars plana vitrectomy (PPV) with internal limiting membrane (ILM) peeling, an alternative therapeutic strategy, with those of medical treatment for chronic macular edema. We conducted a review of the literature on the microscopic, anatomical, and functional reasons for performing PPV with ILM peeling in patients with diabetic macular edema (DME). We searched the PubMed database for articles published between 2000 and 2017. We used the medical subject heading ?vitrectomy diabetic macular edema? and the keywords ?diabetic macular edema?, ?internal limiting membrane peeling?, ?pars plana vitrectomy?, ?diabetic retinopathy?, and ?optical coherence tomography?. Analysis of the literature revealed that cytokines, vascular endothelial growth factor, reactive oxygen species (ROS), and advanced glycation end-products (AGEs) play a unique role in DME. The vitreous cavity serves as a physiological reservoir for all inflammatory molecules. AGE receptors are localized at the footplates of Müller cells and the external limiting membrane (ELM). The footplates of Müller cells are in contact with the ILM, which suggests that they might be responsible for the structural damage (i.e., thickening) observed in the ILM of patients with DME. Therefore, PPV could allow a reduction of cytokines and pro-inflammatory molecules from the vitreous cavity. ILM peeling could eliminate not only the physical traction of a thickened structure, but also the natural reservoir of AGEs, ROS, and inflammatory molecules. PPV with ILM peeling is a surgical option that should be considered when treating patients with chronic DME. Twit Text FOAVip Surgical Versus Medical Treatment for Diabetic Macular Edema: A Review ????Med Hypothesis Discov Innov Ophthalmol http://www.kidney.de/pget.php?&tw=1&pmid=29560368 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29560368 #FOAvip English Wikipedia <ref>{{Cite pmid|29560368}}</ref> Surgical Versus Medical Treatment for Diabetic Macular Edema: A Review #MMPMID29560368 CRIM N; VELEZ-MONTOYA R; MORALES-CANTON V Med Hypothesis Discov Innov Ophthalmol 2017[Win]; 6 (4): 136-42 PMID29560368show ga We aimed to compare the results of pars plana vitrectomy (PPV) with internal limiting membrane (ILM) peeling, an alternative therapeutic strategy, with those of medical treatment for chronic macular edema. We conducted a review of the literature on the microscopic, anatomical, and functional reasons for performing PPV with ILM peeling in patients with diabetic macular edema (DME). We searched the PubMed database for articles published between 2000 and 2017. We used the medical subject heading ?vitrectomy diabetic macular edema? and the keywords ?diabetic macular edema?, ?internal limiting membrane peeling?, ?pars plana vitrectomy?, ?diabetic retinopathy?, and ?optical coherence tomography?. Analysis of the literature revealed that cytokines, vascular endothelial growth factor, reactive oxygen species (ROS), and advanced glycation end-products (AGEs) play a unique role in DME. The vitreous cavity serves as a physiological reservoir for all inflammatory molecules. AGE receptors are localized at the footplates of Müller cells and the external limiting membrane (ELM). The footplates of Müller cells are in contact with the ILM, which suggests that they might be responsible for the structural damage (i.e., thickening) observed in the ILM of patients with DME. Therefore, PPV could allow a reduction of cytokines and pro-inflammatory molecules from the vitreous cavity. ILM peeling could eliminate not only the physical traction of a thickened structure, but also the natural reservoir of AGEs, ROS, and inflammatory molecules. PPV with ILM peeling is a surgical option that should be considered when treating patients with chronic DME. äSurgical Versus Medical Treatment for Diabetic Macular Edema: A Review(epmc).pdf DeepDyve Pubget Overpricing