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10.1177/1470320317717831 http://scihub22266oqcxt.onion/10.1177/1470320317717831 C5843880!5843880!28685619     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Renin+Angiotensin+Aldosterone+Syst 2017 ; 18 (3): ä Nephropedia Template TP {{tp|p=28685619|t=2017. The mTOR/p70S6K1 signaling pathway in renal fibrosis of children with immunoglobulin A nephropathy |pdf=|usr=}}{{28685619}} gab.com Text The mTOR/p70S6K1 signaling pathway in renal fibrosis of children with immunoglobulin A nephropathy JO: J Renin Angiotensin Aldosterone Syst http://www.kidney.de/pget.php?&tw=1&pmid=28685619 #FOAvip Aim:: The purpose of this study was to explore whether mTOR/p70S6K1 signaling is activated in renal fibrosis of immunoglobulin A nephropathy. Methods:: Seventy-two children with immunoglobulin A nephropathy were divided into three groups according to their clinical features and pathological grades. Six normal renal specimens were included in the control group. The expression levels of angiotensin II, mTOR, p70S6K1, E-cadherin, and ?-smooth muscle actin in renal tissues were determined by immunohistochemistry method, the potential correlations of these indexes and relationship between these indexes and the clinicopathological indexes were analyzed. Results:: Compared to the control group, the expression levels of angiotensin II, mTOR, p70S6K1, and ?-smooth muscle actin were significantly higher and the expression levels of E-cadherin were lower both in glomeruli and tubulointerstitium of immunoglobulin A nephropathy children. And the most significant differences were found in the nephrotic syndrome group and pathological grade IV group. In immunoglobulin A nephropathy renal tissues, the expression levels of angiotensin II in glomeruli and tubulointerstitium were both positively correlated with the expression levels of mTOR and ?- smooth muscle actin, and negatively correlated with the expression levels of E-cadherin. Conclusion:: The mTOR/p70S6K1 signaling was activated in renal tissues of children with immunoglobulin A nephropathy, and future studies will need to address the mechanism of mTOR/p70S6K1 signaling in the progress of renal fibrosis in immunoglobulin A nephropathy. Twit Text FOAVip The mTOR/p70S6K1 signaling pathway in renal fibrosis of children with immunoglobulin A nephropathy ????J Renin Angiotensin Aldosterone Syst http://www.kidney.de/pget.php?&tw=1&pmid=28685619 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28685619 #FOAvip English Wikipedia <ref>{{Cite pmid|28685619}}</ref> The mTOR/p70S6K1 signaling pathway in renal fibrosis of children with immunoglobulin A nephropathy #MMPMID28685619 Xu Y; Ling Y; Yang F; Deng J; Rong L; Jiang M; Jiang X J Renin Angiotensin Aldosterone Syst 2017[Jul]; 18 (3): ä PMID28685619show ga Aim:: The purpose of this study was to explore whether mTOR/p70S6K1 signaling is activated in renal fibrosis of immunoglobulin A nephropathy. Methods:: Seventy-two children with immunoglobulin A nephropathy were divided into three groups according to their clinical features and pathological grades. Six normal renal specimens were included in the control group. The expression levels of angiotensin II, mTOR, p70S6K1, E-cadherin, and ?-smooth muscle actin in renal tissues were determined by immunohistochemistry method, the potential correlations of these indexes and relationship between these indexes and the clinicopathological indexes were analyzed. Results:: Compared to the control group, the expression levels of angiotensin II, mTOR, p70S6K1, and ?-smooth muscle actin were significantly higher and the expression levels of E-cadherin were lower both in glomeruli and tubulointerstitium of immunoglobulin A nephropathy children. And the most significant differences were found in the nephrotic syndrome group and pathological grade IV group. In immunoglobulin A nephropathy renal tissues, the expression levels of angiotensin II in glomeruli and tubulointerstitium were both positively correlated with the expression levels of mTOR and ?- smooth muscle actin, and negatively correlated with the expression levels of E-cadherin. Conclusion:: The mTOR/p70S6K1 signaling was activated in renal tissues of children with immunoglobulin A nephropathy, and future studies will need to address the mechanism of mTOR/p70S6K1 signaling in the progress of renal fibrosis in immunoglobulin A nephropathy. äThe mTOR/p70S6K1 signaling pathway in renal fibrosis of children with (epmc).pdf DeepDyve Pubget Overpricing