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10.1097/MD.0000000000010004 http://scihub22266oqcxt.onion/10.1097/MD.0000000000010004 C5842025!5842025 !29465536     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29465536 &cmd=llinks): Failed to open stream: HTTP request failed! 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Primary cutaneous amyloidosis associated with autoimmune hepatitis-primary biliary cirrhosis overlap syndrome and Sjögren syndrome: A case report |pdf=|usr=}}{{29465536 }} gab.com Text Primary cutaneous amyloidosis associated with autoimmune hepatitis-primary biliary cirrhosis overlap syndrome and Sjögren syndrome: A case report JO: Medicine (Baltimore) http://www.kidney.de/pget.php?&tw=1&pmid=29465536 #FOAvip RATIONALE: Primary cutaneous amyloidosis (PCA) is a localized skin disorder characterized by the abnormal deposition of amyloid in the extracellular matrix of the dermis. The association between PCA and other diseases, although rare, has been documented for various autoimmune diseases. PCA associated with autoimmune hepatitis-primary biliary cirrhosis (AIH-PBC) overlap syndrome and Sjögren syndrome (SS) has not been previously reported in the literature. PATIENT CONCERNS: A 50-year-old woman presented with progressive abnormal liver enzyme levels and was referred to our department. DIAGNOSES: Due to the patient's symptoms, laboratory test results, radiographic findings, and pathologic results, she was diagnosed with PCA associated with AIH-PBC overlap syndrome and SS. INTERVENTIONS: She was subsequently treated with a combination of ursodeoxycholic acid (UDCA), prednisone, and azathioprine. OUTCOMES: While this treatment can achieve therapeutic success, it cannot prevent complications from cirrhosis. This patient remains alive but experienced an emergent gastrointestinal hemorrhage. LESSONS: While we acknowledge that this is a single case, these findings extend our knowledge of immunological diseases associated with PCA and suggest a common, immune-mediated pathogenic pathway between PCA, AIH-PBC overlap syndrome, and SS. After 12 years of follow up, clinical manifestations have developed, and these autoimmune diseases have progressed. The combination of UDCA, prednisone, and azathioprine can achieve therapeutic success but cannot prevent disease progression. Routine follow up for this patient is necessary to document disease progression. Twit Text FOAVip Primary cutaneous amyloidosis associated with autoimmune hepatitis-primary biliary cirrhosis overlap syndrome and Sjögren syndrome: A case report ????Medicine (Baltimore) http://www.kidney.de/pget.php?&tw=1&pmid=29465536 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29465536 #FOAvip English Wikipedia <ref>{{Cite pmid|29465536 }}</ref> Primary cutaneous amyloidosis associated with autoimmune hepatitis-primary biliary cirrhosis overlap syndrome and Sjögren syndrome: A case report #MMPMID29465536 Yan X ; Jin J Medicine (Baltimore) 2018[Feb]; 97 (8 ): e0004 PMID29465536 show ga RATIONALE: Primary cutaneous amyloidosis (PCA) is a localized skin disorder characterized by the abnormal deposition of amyloid in the extracellular matrix of the dermis. The association between PCA and other diseases, although rare, has been documented for various autoimmune diseases. PCA associated with autoimmune hepatitis-primary biliary cirrhosis (AIH-PBC) overlap syndrome and Sjögren syndrome (SS) has not been previously reported in the literature. PATIENT CONCERNS: A 50-year-old woman presented with progressive abnormal liver enzyme levels and was referred to our department. DIAGNOSES: Due to the patient's symptoms, laboratory test results, radiographic findings, and pathologic results, she was diagnosed with PCA associated with AIH-PBC overlap syndrome and SS. INTERVENTIONS: She was subsequently treated with a combination of ursodeoxycholic acid (UDCA), prednisone, and azathioprine. OUTCOMES: While this treatment can achieve therapeutic success, it cannot prevent complications from cirrhosis. This patient remains alive but experienced an emergent gastrointestinal hemorrhage. LESSONS: While we acknowledge that this is a single case, these findings extend our knowledge of immunological diseases associated with PCA and suggest a common, immune-mediated pathogenic pathway between PCA, AIH-PBC overlap syndrome, and SS. After 12 years of follow up, clinical manifestations have developed, and these autoimmune diseases have progressed. The combination of UDCA, prednisone, and azathioprine can achieve therapeutic success but cannot prevent disease progression. Routine follow up for this patient is necessary to document disease progression. |Amyloidosis, Familial/drug therapy/*immunology [MESH] |Anti-Inflammatory Agents/administration & dosage [MESH] |Azathioprine/administration & dosage [MESH] |Cholagogues and Choleretics/administration & dosage [MESH] |Drug Therapy, Combination [MESH] |Female [MESH] |Hepatitis, Autoimmune/*complications/drug therapy/immunology [MESH] |Humans [MESH] |Immunosuppressive Agents/administration & dosage [MESH] |Liver Cirrhosis, Biliary/*complications/drug therapy/immunology [MESH] |Middle Aged [MESH] |Prednisone/administration & dosage [MESH] |Sjogren's Syndrome/*complications/drug therapy/immunology [MESH] |Skin Diseases, Genetic/drug therapy/*immunology [MESH] |Treatment Outcome [MESH] |Undifferentiated Connective Tissue Diseases/*complications/drug therapy/immunology [MESH]Primary cutaneous amyloidosis associated with autoimmune hepatitis-pri(epmc).pdf DeepDyve Pubget Overpricing