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10.5051/jpis.2018.48.1.60 http://scihub22266oqcxt.onion/10.5051/jpis.2018.48.1.60 C5841268!5841268!29535891     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Periodontal+Implant+Sci 2018 ; 48 (1): 60-8 Nephropedia Template TP {{tp|p=29535891|t=2018. Porphyromonas gingivalis accelerates atherosclerosis through oxidation of high-density lipoprotein |pdf=|usr=}}{{29535891}} gab.com Text Porphyromonas gingivalis accelerates atherosclerosis through oxidation of high-density lipoprotein JO: J Periodontal Implant Sci http://www.kidney.de/pget.php?&tw=1&pmid=29535891 #FOAvip Purpose: The aim of this study was to evaluate the ability of Porphyromonas gingivalis (P. gingivalis) to induce oxidation of high-density lipoprotein (HDL) and to determine whether the oxidized HDL induced by P. gingivalis exhibited altered antiatherogenic function or became proatherogenic. Methods: P. gingivalis and THP-1 monocytes were cultured, and the extent of HDL oxidation induced by P. gingivalis was evaluated by a thiobarbituric acid-reactive substances (TBARS) assay. To evaluate the altered antiatherogenic and proatherogenic properties of P. gingivalis-treated HDL, lipid oxidation was quantified by the TBARS assay, and tumor necrosis factor alpha (TNF-?) levels and the gelatinolytic activity of matrix metalloproteinase (MMP)-9 were also measured. After incubating macrophages with HDL and P. gingivalis, Oil Red O staining was performed to examine foam cells. Results: P. gingivalis induced HDL oxidation. The HDL treated by P. gingivalis did not reduce lipid oxidation and may have enhanced the formation of MMP-9 and TNF-?. P. gingivalis-treated macrophages exhibited more lipid aggregates than untreated macrophages. Conclusions: P. gingivalis induced HDL oxidation, impairing the atheroprotective function of HDL and making it proatherogenic by eliciting a proinflammatory response through its interaction with monocytes/macrophages. Twit Text FOAVip Porphyromonas gingivalis accelerates atherosclerosis through oxidation of high-density lipoprotein ????J Periodontal Implant Sci http://www.kidney.de/pget.php?&tw=1&pmid=29535891 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29535891 #FOAvip English Wikipedia <ref>{{Cite pmid|29535891}}</ref> Porphyromonas gingivalis accelerates atherosclerosis through oxidation of high-density lipoprotein #MMPMID29535891 Kim HJ; Cha GS; Kim HJ; Kwon EY; Lee JY; Choi J; Joo JY J Periodontal Implant Sci 2018[Feb]; 48 (1): 60-8 PMID29535891show ga Purpose: The aim of this study was to evaluate the ability of Porphyromonas gingivalis (P. gingivalis) to induce oxidation of high-density lipoprotein (HDL) and to determine whether the oxidized HDL induced by P. gingivalis exhibited altered antiatherogenic function or became proatherogenic. Methods: P. gingivalis and THP-1 monocytes were cultured, and the extent of HDL oxidation induced by P. gingivalis was evaluated by a thiobarbituric acid-reactive substances (TBARS) assay. To evaluate the altered antiatherogenic and proatherogenic properties of P. gingivalis-treated HDL, lipid oxidation was quantified by the TBARS assay, and tumor necrosis factor alpha (TNF-?) levels and the gelatinolytic activity of matrix metalloproteinase (MMP)-9 were also measured. After incubating macrophages with HDL and P. gingivalis, Oil Red O staining was performed to examine foam cells. Results: P. gingivalis induced HDL oxidation. The HDL treated by P. gingivalis did not reduce lipid oxidation and may have enhanced the formation of MMP-9 and TNF-?. P. gingivalis-treated macrophages exhibited more lipid aggregates than untreated macrophages. Conclusions: P. gingivalis induced HDL oxidation, impairing the atheroprotective function of HDL and making it proatherogenic by eliciting a proinflammatory response through its interaction with monocytes/macrophages. äPorphyromonas gingivalis accelerates atherosclerosis through oxidation(epmc).pdf DeepDyve Pubget Overpricing