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10.1093/ckj/sfs013 http://scihub22266oqcxt.onion/10.1093/ckj/sfs013 C5783213!5783213!29497527     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Kidney+J 2012 ; 5 (2): 187-91 Nephropedia Template TP {{tp|p=29497527|t=2012. The meaning of the blood urea nitrogen/creatinine ratio in acute kidney injury |pdf=|usr=}}{{29497527}} gab.com Text The meaning of the blood urea nitrogen/creatinine ratio in acute kidney injury JO: Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=29497527 #FOAvip Background.A blood urea nitrogen (BUN)/creatinine ratio (BCR) >20 (0.081 in international unit) is used to distinguish pre-renal azotemia (PRA) and acute tubular necrosis (ATN). However, there is little evidence that BCR can distinguish between these two conditions and/or is clinically useful. Methods.We conducted a retrospective study using a large hospital database. Patients were divided into three groups: ?low BCR? (if BCR when acute kidney injury (AKI) developed was ?20), ?high BCR? (if BCR when AKI developed was >20) and ?no AKI? if patients did not satisfy any of the Risk, Injury, Failure, Loss and End-stage kidney disease criteria for AKI during hospitalization. Results.Among 20 126 study patients, 3641 (18.1%) had AKI. Among these patients, 1704 (46.8%) had a BCR <20 at AKI diagnosis (?low BCR?) and 1937 (53.2%) had a BCR >20 (?high BCR?). The average BCR for the two groups was 15.8 versus 26.1 (P < 0.001). Hospital mortality was significantly less in the ?low-BCR? group (18.4 versus 29.9%, P < 0.001). Multivariable logistic regression analysis for hospital mortality (?no AKI? as a reference) showed that the odds ratio of ?high BCR? (5.73) was higher than that of ?low BCR? (3.32). Conclusions.Approximately half of the patients with AKI have a BCR >20, the traditional threshold of diagnosing PRA. Unlike PRA patients who have a lower mortality than ATN patients, high BCR patients had higher hospital mortality compared with low BCR patients, which was confirmed with multivariable analysis. These findings do not support BCR as a marker of PRA. Twit Text FOAVip The meaning of the blood urea nitrogen/creatinine ratio in acute kidney injury ????Clin Kidney J http://www.kidney.de/pget.php?&tw=1&pmid=29497527 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29497527 #FOAvip English Wikipedia <ref>{{Cite pmid|29497527}}</ref> The meaning of the blood urea nitrogen/creatinine ratio in acute kidney injury #MMPMID29497527 Uchino S; Bellomo R; Goldsmith D Clin Kidney J 2012[Apr]; 5 (2): 187-91 PMID29497527show ga Background.A blood urea nitrogen (BUN)/creatinine ratio (BCR) >20 (0.081 in international unit) is used to distinguish pre-renal azotemia (PRA) and acute tubular necrosis (ATN). However, there is little evidence that BCR can distinguish between these two conditions and/or is clinically useful. Methods.We conducted a retrospective study using a large hospital database. Patients were divided into three groups: ?low BCR? (if BCR when acute kidney injury (AKI) developed was ?20), ?high BCR? (if BCR when AKI developed was >20) and ?no AKI? if patients did not satisfy any of the Risk, Injury, Failure, Loss and End-stage kidney disease criteria for AKI during hospitalization. Results.Among 20 126 study patients, 3641 (18.1%) had AKI. Among these patients, 1704 (46.8%) had a BCR <20 at AKI diagnosis (?low BCR?) and 1937 (53.2%) had a BCR >20 (?high BCR?). The average BCR for the two groups was 15.8 versus 26.1 (P < 0.001). Hospital mortality was significantly less in the ?low-BCR? group (18.4 versus 29.9%, P < 0.001). Multivariable logistic regression analysis for hospital mortality (?no AKI? as a reference) showed that the odds ratio of ?high BCR? (5.73) was higher than that of ?low BCR? (3.32). Conclusions.Approximately half of the patients with AKI have a BCR >20, the traditional threshold of diagnosing PRA. Unlike PRA patients who have a lower mortality than ATN patients, high BCR patients had higher hospital mortality compared with low BCR patients, which was confirmed with multivariable analysis. These findings do not support BCR as a marker of PRA. äThe meaning of the blood urea nitrogen/creatinine ratio in acute kidne(epmc).pdf DeepDyve Pubget Overpricing