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10.18240/ijo.2017.11.19 http://scihub22266oqcxt.onion/10.18240/ijo.2017.11.19 C5686377!5686377 !29181322     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29181322 &cmd=llinks): Failed to open stream: HTTP request failed! 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Potential role of Müller cells in the pathogenesis of macropsia associated with epiretinal membrane: a hypothesis revisited |pdf=|usr=}}{{29181322 }} gab.com Text Potential role of Müller cells in the pathogenesis of macropsia associated with epiretinal membrane: a hypothesis revisited JO: Int J Ophthalmol http://www.kidney.de/pget.php?&tw=1&pmid=29181322 #FOAvip Pathophysiological explanations for metamorphopsia associated with retinal pathologies generally focus on photoreceptor organization disruption. However, the retinal microarchitecture is complicated, and we hypothesize that other retinal cells may also be involved. Metamorphopsia has been widely studied in eyes with epiretinal membranes and we revisit the idea that Müller cell displacement causes retinal macropsia. A PubMed query and related article search for the macula ultrastructure under normal and pathological conditions revealed an enormous amount of information, particularly ultrahigh definition optical coherence tomography and other retinal imaging modality studies. Findings of these imaging studies support our hypothesis that Müller cells, and not cone photoreceptors, are primarily responsible for macropsia in eyes with epiretinal membranes. More specifically, we conclude that displacement of Müller cell endfeet, and not photoreceptor cones, is a more likely the explanation for retinal macropsia associated with epiretinal membranes. Twit Text FOAVip Potential role of Müller cells in the pathogenesis of macropsia associated with epiretinal membrane: a hypothesis revisited ????Int J Ophthalmol http://www.kidney.de/pget.php?&tw=1&pmid=29181322 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29181322 #FOAvip English Wikipedia <ref>{{Cite pmid|29181322 }}</ref> Potential role of Müller cells in the pathogenesis of macropsia associated with epiretinal membrane: a hypothesis revisited #MMPMID29181322 Colakoglu A ; Balci Akar S Int J Ophthalmol 2017[]; 10 (11 ): 1759-1767 PMID29181322 show ga Pathophysiological explanations for metamorphopsia associated with retinal pathologies generally focus on photoreceptor organization disruption. However, the retinal microarchitecture is complicated, and we hypothesize that other retinal cells may also be involved. Metamorphopsia has been widely studied in eyes with epiretinal membranes and we revisit the idea that Müller cell displacement causes retinal macropsia. A PubMed query and related article search for the macula ultrastructure under normal and pathological conditions revealed an enormous amount of information, particularly ultrahigh definition optical coherence tomography and other retinal imaging modality studies. Findings of these imaging studies support our hypothesis that Müller cells, and not cone photoreceptors, are primarily responsible for macropsia in eyes with epiretinal membranes. More specifically, we conclude that displacement of Müller cell endfeet, and not photoreceptor cones, is a more likely the explanation for retinal macropsia associated with epiretinal membranes. äPotential role of Müller cells in the pathogenesis of macropsia associ(epmc).pdf DeepDyve Pubget Overpricing