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10.1186/s13256-017-1449-2 http://scihub22266oqcxt.onion/10.1186/s13256-017-1449-2 C5684746!5684746 !29132419     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29132419 &cmd=llinks): Failed to open stream: HTTP request failed! 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Success of anti-CD20 monoclonal antibody treatment for severe autoimmune hemolytic anemia caused by warm-reactive immunoglobulin A, immunoglobulin G, and immunoglobulin M autoantibodies in a child: a case report |pdf=|usr=}}{{29132419 }} gab.com Text Success of anti-CD20 monoclonal antibody treatment for severe autoimmune hemolytic anemia caused by warm-reactive immunoglobulin A, immunoglobulin G, and immunoglobulin M autoantibodies in a child: a case report JO: J Med Case Rep http://www.kidney.de/pget.php?&tw=1&pmid=29132419 #FOAvip BACKGROUND: Autoimmune hemolytic anemia is rare in children. First-line therapies for this disease consist of corticosteroids and intravenously administered immunoglobulin that are effective in most patients. However, a small proportion of cases (5 to 10%) is refractory to these therapies and may represent a medical emergency, especially when hemolysis is due to warm immunoglobulin M. Recently, reports of the use of rituximab in adult autoimmune diseases have shown promising results. In children, there are few studies on the use of rituximab in the treatment for autoimmune hemolytic anemia, especially on its long-term efficacy and adverse effects. CASE PRESENTATION: Here, we report the case of a 10-year-old Tunisian girl with refractory acute autoimmune hemolytic anemia caused by warm-reactive immunoglobulin A, immunoglobulin G, immunoglobulin M, and C3d autoantibodies. First-line treatments using corticosteroids and intravenously administered immunoglobulin were ineffective in controlling her severe disease. On the other hand, she was successfully treated with rituximab. In fact, her hemolytic anemia improved rapidly and no adverse effects were observed. CONCLUSIONS: The case that we report in this paper shows that rituximab could be an alternative therapeutic option in severe acute autoimmune hemolytic anemia with profound hemolysis refractory to conventional treatment. Moreover, it may preclude the use of plasmapheresis in such an urgent situation with a sustained remission. Twit Text FOAVip Success of anti-CD20 monoclonal antibody treatment for severe autoimmune hemolytic anemia caused by warm-reactive immunoglobulin A, immunoglobulin G, and immunoglobulin M autoantibodies in a child: a case report ????J Med Case Rep http://www.kidney.de/pget.php?&tw=1&pmid=29132419 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29132419 #FOAvip English Wikipedia <ref>{{Cite pmid|29132419 }}</ref> Success of anti-CD20 monoclonal antibody treatment for severe autoimmune hemolytic anemia caused by warm-reactive immunoglobulin A, immunoglobulin G, and immunoglobulin M autoantibodies in a child: a case report #MMPMID29132419 Ajmi H ; Mabrouk S ; Hassayoun S ; Regaieg H ; Tfifha M ; Jalel C ; Skouri H ; Zouari N ; Abroug S J Med Case Rep 2017[Nov]; 11 (1 ): 321 PMID29132419 show ga BACKGROUND: Autoimmune hemolytic anemia is rare in children. First-line therapies for this disease consist of corticosteroids and intravenously administered immunoglobulin that are effective in most patients. However, a small proportion of cases (5 to 10%) is refractory to these therapies and may represent a medical emergency, especially when hemolysis is due to warm immunoglobulin M. Recently, reports of the use of rituximab in adult autoimmune diseases have shown promising results. In children, there are few studies on the use of rituximab in the treatment for autoimmune hemolytic anemia, especially on its long-term efficacy and adverse effects. CASE PRESENTATION: Here, we report the case of a 10-year-old Tunisian girl with refractory acute autoimmune hemolytic anemia caused by warm-reactive immunoglobulin A, immunoglobulin G, immunoglobulin M, and C3d autoantibodies. First-line treatments using corticosteroids and intravenously administered immunoglobulin were ineffective in controlling her severe disease. On the other hand, she was successfully treated with rituximab. In fact, her hemolytic anemia improved rapidly and no adverse effects were observed. CONCLUSIONS: The case that we report in this paper shows that rituximab could be an alternative therapeutic option in severe acute autoimmune hemolytic anemia with profound hemolysis refractory to conventional treatment. Moreover, it may preclude the use of plasmapheresis in such an urgent situation with a sustained remission. |Anemia, Hemolytic, Autoimmune/*chemically induced/diagnosis/*drug therapy [MESH] |Antigens, CD20 [MESH] |Blood Transfusion [MESH] |Child [MESH] |Female [MESH] |Humans [MESH] |Immunoglobulin A/administration & dosage/adverse effects [MESH] |Immunoglobulin G/administration & dosage/adverse effects [MESH] |Immunoglobulin M/administration & dosage/adverse effects [MESH] |Immunoglobulins, Intravenous/administration & dosage/*adverse effects [MESH] |Immunologic Factors/*administration & dosage/adverse effects [MESH] |Leukocyte Count [MESH] |Remission Induction [MESH] |Rituximab/*administration & dosage/adverse effects [MESH]Success of anti-CD20 monoclonal antibody treatment for severe autoimmu(epmc).pdf DeepDyve Pubget Overpricing