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10.21037/tp.2017.09.04 http://scihub22266oqcxt.onion/10.21037/tp.2017.09.04 C5682385!5682385!29184809     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Transl+Pediatr 2017 ; 6 (4): 274-85 Nephropedia Template TP {{tp|p=29184809|t=2017. Review of Prader-Willi syndrome: the endocrine approach |pdf=|usr=}}{{29184809}} gab.com Text Review of Prader-Willi syndrome: the endocrine approach JO: Transl Pediatr http://www.kidney.de/pget.php?&tw=1&pmid=29184809 #FOAvip Prader-Willi syndrome (PWS) is a complex genetic disorder with implications on the endocrine and neurologic systems, metabolism, and behavior. Early in life, PWS is characterized by hypotonia and failure to thrive, followed by obesity and hyperphagia. Patients with PWS develop hypothalamic dysfunction which may lead growth hormone deficiency (GHD), hypogonadism, hypothyroidism, adrenal insufficiency, and poor bone mineral density (BMD). In addition to hypothalamic dysfunction, individuals with PWS have increased risk for obesity which may be complicated by metabolic syndrome and type 2 diabetes mellitus (T2DM). In this paper, we will review the current literature pertaining to the endocrine concerns of PWS and current recommendations for screening and management of these conditions. Twit Text FOAVip Review of Prader-Willi syndrome: the endocrine approach ????Transl Pediatr http://www.kidney.de/pget.php?&tw=1&pmid=29184809 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29184809 #FOAvip English Wikipedia <ref>{{Cite pmid|29184809}}</ref> Review of Prader-Willi syndrome: the endocrine approach #MMPMID29184809 Heksch R; Kamboj M; Anglin K; Obrynba K Transl Pediatr 2017[Oct]; 6 (4): 274-85 PMID29184809show ga Prader-Willi syndrome (PWS) is a complex genetic disorder with implications on the endocrine and neurologic systems, metabolism, and behavior. Early in life, PWS is characterized by hypotonia and failure to thrive, followed by obesity and hyperphagia. Patients with PWS develop hypothalamic dysfunction which may lead growth hormone deficiency (GHD), hypogonadism, hypothyroidism, adrenal insufficiency, and poor bone mineral density (BMD). In addition to hypothalamic dysfunction, individuals with PWS have increased risk for obesity which may be complicated by metabolic syndrome and type 2 diabetes mellitus (T2DM). In this paper, we will review the current literature pertaining to the endocrine concerns of PWS and current recommendations for screening and management of these conditions. äReview of Prader-Willi syndrome: the endocrine approach (epmc).pdf DeepDyve Pubget Overpricing