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10.3892/or.2017.5923 http://scihub22266oqcxt.onion/10.3892/or.2017.5923 C5652940!5652940 !28849076     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28849076 &cmd=llinks): Failed to open stream: HTTP request failed! 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Upregulation of GNL3 expression promotes colon cancer cell proliferation, migration, invasion and epithelial-mesenchymal transition via the Wnt/?-catenin signaling pathway |pdf=|usr=}}{{28849076 }} gab.com Text Upregulation of GNL3 expression promotes colon cancer cell proliferation, migration, invasion and epithelial-mesenchymal transition via the Wnt/ -catenin signaling pathway JO: Oncol Rep http://www.kidney.de/pget.php?&tw=1&pmid=28849076 #FOAvip G protein nucleolar 3 (GNL3), a nucleolar GTP-binding protein, is highly expressed in progenitor cells, stem cells, and various types of cancer cells. Therefore, it is considered to have an important role in cancer pathogenesis. GNL3 has been reported to play crucial roles in cell proliferation, cell cycle regulation, inhibition of differentiation, ribosome biogenesis, and the maintenance of stemness, genome stability and telomere integrity. Furthermore, GNL3 has recently been shown to be involved in cancer invasion and metastasis. However, the biological significance of GNL3 in the invasion and metastasis of colon cancer remains unclear. This study was performed to address this gap in knowledge. GNL3 expression was upregulated in colon cancer tissue specimens and correlated with tumor differentiation, invasion and metastasis. GNL3 overexpression promoted cell proliferation, invasion, migration and the epithelial-mesenchymal transition (EMT) in colon cancer cells. Moreover, inhibition of the EMT and the Wnt/??catenin signaling pathway induced by GNL3 knockdown was partially reversed by lithium chloride (LiCl). Based on these data, GNL3 promotes the EMT in colon cancer by activating the Wnt/??catenin signaling pathway. In summary, GNL3 is upregulated in colon cancer and plays an important role in tumor growth, invasion and metastasis. Strategies targeting GNL3 are potential treatments for colon cancer. Twit Text FOAVip Upregulation of GNL3 expression promotes colon cancer cell proliferation, migration, invasion and epithelial-mesenchymal transition via the Wnt/ -catenin signaling pathway ????Oncol Rep http://www.kidney.de/pget.php?&tw=1&pmid=28849076 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28849076 #FOAvip English Wikipedia <ref>{{Cite pmid|28849076 }}</ref> Upregulation of GNL3 expression promotes colon cancer cell proliferation, migration, invasion and epithelial-mesenchymal transition via the Wnt/?-catenin signaling pathway #MMPMID28849076 Tang X ; Zha L ; Li H ; Liao G ; Huang Z ; Peng X ; Wang Z Oncol Rep 2017[Oct]; 38 (4 ): 2023-2032 PMID28849076 show ga G protein nucleolar 3 (GNL3), a nucleolar GTP-binding protein, is highly expressed in progenitor cells, stem cells, and various types of cancer cells. Therefore, it is considered to have an important role in cancer pathogenesis. GNL3 has been reported to play crucial roles in cell proliferation, cell cycle regulation, inhibition of differentiation, ribosome biogenesis, and the maintenance of stemness, genome stability and telomere integrity. Furthermore, GNL3 has recently been shown to be involved in cancer invasion and metastasis. However, the biological significance of GNL3 in the invasion and metastasis of colon cancer remains unclear. This study was performed to address this gap in knowledge. GNL3 expression was upregulated in colon cancer tissue specimens and correlated with tumor differentiation, invasion and metastasis. GNL3 overexpression promoted cell proliferation, invasion, migration and the epithelial-mesenchymal transition (EMT) in colon cancer cells. Moreover, inhibition of the EMT and the Wnt/??catenin signaling pathway induced by GNL3 knockdown was partially reversed by lithium chloride (LiCl). Based on these data, GNL3 promotes the EMT in colon cancer by activating the Wnt/??catenin signaling pathway. In summary, GNL3 is upregulated in colon cancer and plays an important role in tumor growth, invasion and metastasis. Strategies targeting GNL3 are potential treatments for colon cancer. |*Wnt Signaling Pathway [MESH] |Caco-2 Cells [MESH] |Cell Line, Tumor [MESH] |Cell Movement/physiology [MESH] |Cell Proliferation/physiology [MESH] |Colonic Neoplasms/*metabolism/pathology [MESH] |Epithelial-Mesenchymal Transition [MESH] |GTP-Binding Proteins/*biosynthesis [MESH] |HCT116 Cells [MESH] |HT29 Cells [MESH] |Humans [MESH] |Neoplasm Invasiveness [MESH] |Nuclear Proteins/*biosynthesis [MESH] |Up-Regulation [MESH]Upregulation of GNL3 expression promotes colon cancer cell proliferati(epmc).pdf DeepDyve Pubget Overpricing