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10.1016/j.celrep.2017.09.006 http://scihub22266oqcxt.onion/10.1016/j.celrep.2017.09.006 C5621787!5621787 !28954233     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28954233 &cmd=llinks): Failed to open stream: HTTP request failed! 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Lymphatic Vessels Balance Viral Dissemination and Immune Activation following Cutaneous Viral Infection |pdf=|usr=}}{{28954233 }} gab.com Text Lymphatic Vessels Balance Viral Dissemination and Immune Activation following Cutaneous Viral Infection JO: Cell Rep http://www.kidney.de/pget.php?&tw=1&pmid=28954233 #FOAvip Lymphatic vessels lie at the interface between peripheral sites of pathogen entry, adaptive immunity, and the systemic host. Though the paradigm is that their open structure allows for passive flow of infectious particles from peripheral tissues to lymphoid organs, virus applied to skin by scarification does not spread to draining lymph nodes. Using cutaneous infection by scarification, we analyzed the effect of viral infection on lymphatic transport and evaluated its role at the host-pathogen interface. We found that, in the absence of lymphatic vessels, canonical lymph-node-dependent immune induction was impaired, resulting in exacerbated pathology and compensatory, systemic priming. Furthermore, lymphatic vessels decouple fluid and cellular transport in an interferon-dependent manner, leading to viral sequestration while maintaining dendritic cell transport for immune induction. In conclusion, we found that lymphatic vessels balance immune activation and viral dissemination and act as an "innate-like" component of tissue host viral defense. Twit Text FOAVip Lymphatic Vessels Balance Viral Dissemination and Immune Activation following Cutaneous Viral Infection ????Cell Rep http://www.kidney.de/pget.php?&tw=1&pmid=28954233 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28954233 #FOAvip English Wikipedia <ref>{{Cite pmid|28954233 }}</ref> Lymphatic Vessels Balance Viral Dissemination and Immune Activation following Cutaneous Viral Infection #MMPMID28954233 Loo CP ; Nelson NA ; Lane RS ; Booth JL ; Loprinzi Hardin SC ; Thomas A ; Slifka MK ; Nolz JC ; Lund AW Cell Rep 2017[Sep]; 20 (13 ): 3176-3187 PMID28954233 show ga Lymphatic vessels lie at the interface between peripheral sites of pathogen entry, adaptive immunity, and the systemic host. Though the paradigm is that their open structure allows for passive flow of infectious particles from peripheral tissues to lymphoid organs, virus applied to skin by scarification does not spread to draining lymph nodes. Using cutaneous infection by scarification, we analyzed the effect of viral infection on lymphatic transport and evaluated its role at the host-pathogen interface. We found that, in the absence of lymphatic vessels, canonical lymph-node-dependent immune induction was impaired, resulting in exacerbated pathology and compensatory, systemic priming. Furthermore, lymphatic vessels decouple fluid and cellular transport in an interferon-dependent manner, leading to viral sequestration while maintaining dendritic cell transport for immune induction. In conclusion, we found that lymphatic vessels balance immune activation and viral dissemination and act as an "innate-like" component of tissue host viral defense. |Animals [MESH] |Humans [MESH] |Lymph Nodes/immunology [MESH] |Lymphatic System/*virology [MESH] |Lymphatic Vessels/*virology [MESH] |Mice [MESH]Lymphatic Vessels Balance Viral Dissemination and Immune Activation fo(epmc).pdf DeepDyve Pubget Overpricing