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10.1016/j.toxrep.2016.07.005 http://scihub22266oqcxt.onion/10.1016/j.toxrep.2016.07.005 C5616016!5616016 !28959582     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28959582 &cmd=llinks): Failed to open stream: HTTP request failed! 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Calcium, zinc and vitamin E ameliorate cadmium-induced renal oxidative damage in albino Wistar rats |pdf=|usr=}}{{28959582 }} gab.com Text Calcium, zinc and vitamin E ameliorate cadmium-induced renal oxidative damage in albino Wistar rats JO: Toxicol Rep http://www.kidney.de/pget.php?&tw=1&pmid=28959582 #FOAvip This study was aimed to examine the protective effects of supplementation with calcium + zinc (Ca + Zn) or vitamin E (Vit-E) on Cd-induced renal oxidative damage. Young albino Wistar rats (180 ± 10 g) (n = 6) control rats, Cd, Cd + Ca + Zn, and Cd + Vit-E experimental groups and the experimental period was 30 days. Rats were exposed to Cd (20 mg/kg body weight) alone treated as Cd treated group and the absence or presence of Ca + Zn (2 mg/kg each) or Vit-E (20 mg/kg body weight) supplementation treated as two separate groups. The activities of the stress marker enzymes superoxide dismutase (SOD), catalase (CAT), glutathione reductase (GR), glutathione peroxidase (GPx), glutathione-S-transferase (GST) and lipid peroxidase (LPx) were determined in renal mitochondrial fractions of experimental rats. We observed quantitative changes in SOD isoenzymatic patterns by non-denaturing PAGE analysis, and quantified band densities. These results showed that Cd exposure leads to decreases in SOD, CAT, GR, and GPx activities and a concomitant increase in LPx and GST activities. Ca + Zn and Vit-E administration with Cd significantly reversed Cd-induced perturbations in oxidative stress marker enzymes. However, Vit-E showed more inhibitory activity against Cd than did Ca + Zn, and it protected against Cd-induced nephrotoxicity. Twit Text FOAVip Calcium, zinc and vitamin E ameliorate cadmium-induced renal oxidative damage in albino Wistar rats ????Toxicol Rep http://www.kidney.de/pget.php?&tw=1&pmid=28959582 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28959582 #FOAvip English Wikipedia <ref>{{Cite pmid|28959582 }}</ref> Calcium, zinc and vitamin E ameliorate cadmium-induced renal oxidative damage in albino Wistar rats #MMPMID28959582 Adi PJ ; Burra SP ; Vataparti AR ; Matcha B Toxicol Rep 2016[]; 3 (ä): 591-597 PMID28959582 show ga This study was aimed to examine the protective effects of supplementation with calcium + zinc (Ca + Zn) or vitamin E (Vit-E) on Cd-induced renal oxidative damage. Young albino Wistar rats (180 ± 10 g) (n = 6) control rats, Cd, Cd + Ca + Zn, and Cd + Vit-E experimental groups and the experimental period was 30 days. Rats were exposed to Cd (20 mg/kg body weight) alone treated as Cd treated group and the absence or presence of Ca + Zn (2 mg/kg each) or Vit-E (20 mg/kg body weight) supplementation treated as two separate groups. The activities of the stress marker enzymes superoxide dismutase (SOD), catalase (CAT), glutathione reductase (GR), glutathione peroxidase (GPx), glutathione-S-transferase (GST) and lipid peroxidase (LPx) were determined in renal mitochondrial fractions of experimental rats. We observed quantitative changes in SOD isoenzymatic patterns by non-denaturing PAGE analysis, and quantified band densities. These results showed that Cd exposure leads to decreases in SOD, CAT, GR, and GPx activities and a concomitant increase in LPx and GST activities. Ca + Zn and Vit-E administration with Cd significantly reversed Cd-induced perturbations in oxidative stress marker enzymes. However, Vit-E showed more inhibitory activity against Cd than did Ca + Zn, and it protected against Cd-induced nephrotoxicity. äCalcium, zinc and vitamin E ameliorate cadmium-induced renal oxidative(epmc).pdf DeepDyve Pubget Overpricing