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10.1038/nrgastro.2016.76 http://scihub22266oqcxt.onion/10.1038/nrgastro.2016.76 C5605144!5605144!27251213     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Nat+Rev+Gastroenterol+Hepatol 2016 ; 13 (7): 389-401 Nephropedia Template TP {{tp|p=27251213|t=2016. Vagal neurocircuitry and its influence on gastric motility |pdf=|usr=}}{{27251213}} gab.com Text Vagal neurocircuitry and its influence on gastric motility JO: Nat Rev Gastroenterol Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=27251213 #FOAvip A large body of research has been dedicated to the effects of gastrointestinal peptides on vagal afferent fibres, yet multiple lines of evidence indicate that gastrointestinal peptides also modulate brainstem vagal neurocircuitry, and that this modulation has a fundamental role in the physiology and pathophysiology of the upper gastrointestinal tract. In fact, brainstem vagovagal neurocircuits comprise highly plastic neurons and synapses connecting afferent vagal fibres, second order neurons of the nucleus tractus solitarius (NTS), and efferent fibres originating in the dorsal motor nucleus of the vagus (DMV). Neuronal communication between the NTS and DMV is regulated by the presence of a variety of inputs, both from within the brainstem itself as well as from higher centres, which utilize an array of neurotransmitters and neuromodulators. Because of the circumventricular nature of these brainstem areas, circulating hormones can also modulate the vagal output to the upper gastrointestinal tract. This Review summarizes the organization and function of vagovagal reflex control of the upper gastrointestinal tract, presents data on the plasticity within these neurocircuits after stress, and discusses the gastrointestinal dysfunctions observed in Parkinson disease as examples of physiological adjustment and maladaptation of these reflexes. Twit Text FOAVip Vagal neurocircuitry and its influence on gastric motility ????Nat Rev Gastroenterol Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=27251213 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27251213 #FOAvip English Wikipedia <ref>{{Cite pmid|27251213}}</ref> Vagal neurocircuitry and its influence on gastric motility #MMPMID27251213 Travagli RA; Anselmi L Nat Rev Gastroenterol Hepatol 2016[Jul]; 13 (7): 389-401 PMID27251213show ga A large body of research has been dedicated to the effects of gastrointestinal peptides on vagal afferent fibres, yet multiple lines of evidence indicate that gastrointestinal peptides also modulate brainstem vagal neurocircuitry, and that this modulation has a fundamental role in the physiology and pathophysiology of the upper gastrointestinal tract. In fact, brainstem vagovagal neurocircuits comprise highly plastic neurons and synapses connecting afferent vagal fibres, second order neurons of the nucleus tractus solitarius (NTS), and efferent fibres originating in the dorsal motor nucleus of the vagus (DMV). Neuronal communication between the NTS and DMV is regulated by the presence of a variety of inputs, both from within the brainstem itself as well as from higher centres, which utilize an array of neurotransmitters and neuromodulators. Because of the circumventricular nature of these brainstem areas, circulating hormones can also modulate the vagal output to the upper gastrointestinal tract. This Review summarizes the organization and function of vagovagal reflex control of the upper gastrointestinal tract, presents data on the plasticity within these neurocircuits after stress, and discusses the gastrointestinal dysfunctions observed in Parkinson disease as examples of physiological adjustment and maladaptation of these reflexes. äVagal neurocircuitry and its influence on gastric motility (epmc).pdf DeepDyve Pubget Overpricing