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10.1073/pnas.1705338114 http://scihub22266oqcxt.onion/10.1073/pnas.1705338114 C5604011!5604011!28851829     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28851829&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Proc+Natl+Acad+Sci+U+S+A 2017 ; 114 (37): 9948-53 Nephropedia Template TP {{tp|p=28851829|t=2017. Glucocorticoids promote Von Hippel Lindau degradation and Hif-1? stabilization |pdf=|usr=}}{{28851829}} gab.com Text Glucocorticoids promote Von Hippel Lindau degradation and Hif-1 stabilization JO: Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=28851829 #FOAvip An in vivo chemical screen in zebrafish identified glucocorticoids (GCs) as activators of hypoxia-inducible factor transcriptional responses in the liver. This cross-talk is conserved in human liver and requires glucocorticoid receptor signaling but not DNA binding. In human liver cells, GCs down-regulate Von Hippel Lindau expression at a posttranscriptional level most likely through c-src?mediated proteasomal degradation. Since the liver is an important regulator of blood glucose and hypoxia-inducible factors regulate gluconeogenesis/glycogen synthesis, cross-talk between these transcriptional regulators may be essential to control glucose metabolism in the liver. This identified, conserved, noncanonical pathway may have wider physiological significance in health and disease. Twit Text FOAVip Glucocorticoids promote Von Hippel Lindau degradation and Hif-1 stabilization ????Proc Natl Acad Sci U S A http://www.kidney.de/pget.php?&tw=1&pmid=28851829 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28851829 #FOAvip English Wikipedia <ref>{{Cite pmid|28851829}}</ref> Glucocorticoids promote Von Hippel Lindau degradation and Hif-1? stabilization #MMPMID28851829 Vettori A; Greenald D; Wilson GK; Peron M; Facchinello N; Markham E; Sinnakaruppan M; Matthews LC; McKeating JA; Argenton F; van Eeden FJM Proc Natl Acad Sci U S A 2017[Sep]; 114 (37): 9948-53 PMID28851829show ga An in vivo chemical screen in zebrafish identified glucocorticoids (GCs) as activators of hypoxia-inducible factor transcriptional responses in the liver. This cross-talk is conserved in human liver and requires glucocorticoid receptor signaling but not DNA binding. In human liver cells, GCs down-regulate Von Hippel Lindau expression at a posttranscriptional level most likely through c-src?mediated proteasomal degradation. Since the liver is an important regulator of blood glucose and hypoxia-inducible factors regulate gluconeogenesis/glycogen synthesis, cross-talk between these transcriptional regulators may be essential to control glucose metabolism in the liver. This identified, conserved, noncanonical pathway may have wider physiological significance in health and disease. äGlucocorticoids promote Von Hippel Lindau degradation and Hif-1? stabi(epmc).pdf DeepDyve Pubget Overpricing