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10.18632/oncotarget.19738 http://scihub22266oqcxt.onion/10.18632/oncotarget.19738 C5601744!5601744 !28938648     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28938648 &cmd=llinks): Failed to open stream: HTTP request failed! 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A novel lncRNA, LL22NC03-N64E9 1, represses KLF2 transcription through binding with EZH2 in colorectal cancer |pdf=|usr=}}{{28938648 }} gab.com Text A novel lncRNA, LL22NC03-N64E9 1, represses KLF2 transcription through binding with EZH2 in colorectal cancer JO: Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=28938648 #FOAvip Long noncoding RNAs (lncRNA) have been implicated in variety human cancer but their mechanisms of function are mainly undocumented. In the present study, we investigated lncRNAs alteration that contributed to colorectal cancer (CRC) by utilizing TCGA RNA sequencing data and other publicly available lncRNAs expression profiling data. Here, We screened out the CRC-associated lncRNA LL22NC03-N64E9.1, a key regulator of CRC development and progression. We also revealed that knockdown of LL22NC03-N64E9.1 inhibited cell proliferation, colony formation, tumorigenicity and apoptosis promotion, both in vitro and in vivo. Mechanistically, LL22NC03-N64E9.1 repressed underlying target gene KLF2 transcription through binding to EZH2. Furthermore, rescue experiments revealed that LL22NC03-N64E9.1 oncogenic function may partially depend on repressing KLF2. Taken together, our results suggested that LL22NC03-N64E9.1 confered an oncogenic function in human CRC and may serve as a candidate prognostic biomarker and target for new therapies in this deadly disease. Twit Text FOAVip A novel lncRNA, LL22NC03-N64E9 1, represses KLF2 transcription through binding with EZH2 in colorectal cancer ????Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=28938648 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28938648 #FOAvip English Wikipedia <ref>{{Cite pmid|28938648 }}</ref> A novel lncRNA, LL22NC03-N64E9 1, represses KLF2 transcription through binding with EZH2 in colorectal cancer #MMPMID28938648 Lian Y ; Yan C ; Ding J ; Xia R ; Ma Z ; Hui B ; Ji H ; Zhou J ; Wang K Oncotarget 2017[Aug]; 8 (35 ): 59435-59445 PMID28938648 show ga Long noncoding RNAs (lncRNA) have been implicated in variety human cancer but their mechanisms of function are mainly undocumented. In the present study, we investigated lncRNAs alteration that contributed to colorectal cancer (CRC) by utilizing TCGA RNA sequencing data and other publicly available lncRNAs expression profiling data. Here, We screened out the CRC-associated lncRNA LL22NC03-N64E9.1, a key regulator of CRC development and progression. We also revealed that knockdown of LL22NC03-N64E9.1 inhibited cell proliferation, colony formation, tumorigenicity and apoptosis promotion, both in vitro and in vivo. Mechanistically, LL22NC03-N64E9.1 repressed underlying target gene KLF2 transcription through binding to EZH2. Furthermore, rescue experiments revealed that LL22NC03-N64E9.1 oncogenic function may partially depend on repressing KLF2. Taken together, our results suggested that LL22NC03-N64E9.1 confered an oncogenic function in human CRC and may serve as a candidate prognostic biomarker and target for new therapies in this deadly disease. äA novel lncRNA, LL22NC03-N64E9 1, represses KLF2 transcription through(epmc).pdf DeepDyve Pubget Overpricing